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Toxins|February 24, 2022
Botulinum Neurotoxin Chimeras Suppress Stimulation by Capsaicin of Rat Trigeminal Sensory Neurons In Vivo and In VitroCaren Antoniazzi, Mariia Belinskaia, Tomas Zurawski, et al.
Pharmacology & Therapeutics|January 31, 2016
Distinctive role of KV1.1 subunit in the biology and functions of low threshold K(+) channels with implications for neurological diseaseSaak V Ovsepian, Marie LeBerre, Volker Steuber, et al.
International Journal of Molecular Sciences|January 21, 2022
NGF Enhances CGRP Release Evoked by Capsaicin from Rat Trigeminal Neurons: Differential Inhibition by SNAP-25-Cleaving ProteasesMariia Belinskaia, Tomas Zurawski, Seshu Kumar Kaza, et al.
The Journal of Pharmacology and Experimental Therapeutics|October 20, 2006
Two protein trafficking processes at motor nerve endings unveiled by botulinum neurotoxin EGary Lawrence, Jiafu Wang, C K N Kwo Chion, et al.
BJU International|September 6, 2007
Persistence of the synaptosomal-associated protein-25 cleavage product after intradetrusor botulinum toxin A injections in patients with myelomeningocele showing an inadequate response to treatmentHeinrich Schulte-Baukloh, Tomas H Zurawski, Helmut H Knispel, et al.
The Journal of Physiology|January 16, 2013
A defined heteromeric KV1 channel stabilizes the intrinsic pacemaking and regulates the output of deep cerebellar nuclear neurons to thalamic targetsSaak V Ovsepian, Volker Steuber, Marie Le Berre, et al.
The FEBS Journal|May 28, 2014
Fusion of Golgi-derived vesicles mediated by SNAP-25 is essential for sympathetic neuron outgrowth but relatively insensitive to botulinum neurotoxins in vitroGary W Lawrence, Jiafu Wang, Mitchell F Brin, et al.
Brain Structure & Function|February 11, 2015
Internalization and retrograde axonal trafficking of tetanus toxin in motor neurons and trans-synaptic propagation at central synapses exceed those of its C-terminal-binding fragmentsSaak V Ovsepian, Saak V Ovespian, MacDara Bodeker, et al.
The Biochemical Journal|March 1, 2011
Position-dependent attenuation by Kv1.6 of N-type inactivation of Kv1.4-containing channelsAhmed Al-Sabi, Seshu Kaza, Marie Le Berre, et al.
International Journal of Molecular Sciences|January 21, 2023
Bipartite Activation of Sensory Neurons by a TRPA1 Agonist Allyl Isothiocyanate Is Reflected by Complex Ca<sup>2+</sup> Influx and CGRP Release Patterns: Enhancement by NGF and Inhibition with VAMP and SNAP-25 Cleaving Botulinum NeurotoxinsMariia Belinskaia, Jiafu Wang, Seshu Kumar Kaza, et al.
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