J P Herman|L Stinus|M Le Moal - JoVE Visualize

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Brain Research|June 18, 1984

Functional recovery following transplantation of ventral mesencephalic cells in rat subjected to 6-OHDA lesions of the mesolimbic dopaminergic neuronsD Nadaud, J P Herman, H Simon, et al.

Physiology & Behavior|December 1, 1984

Contributions of the mesencephalic dopaminergic system and the trigeminal sensory pathway to the ventral tegmental aphagia syndrome in ratsD Nadaud, H Simon, J P Herman, et al.

Proceedings of the National Academy of Sciences of the United States of America|April 1, 1980

Locomotor activation induced by infusion of endorphins into the ventral tegmental area: evidence for opiate-dopamine interactionsL Stinus, G F Koob, N Ling, et al.

Brain Research|May 8, 1989

Substance P, neurotensin and enkephalin injections into the ventral tegmental area: comparative study on dopamine turnover in several forebrain structuresM Cador, J M Rivet, A E Kelley, et al.

Brain Research|March 26, 1993

Central administration of corticotropin releasing factor induces long-term sensitization to D-amphetamineM Cador, B J Cole, G F Koob, et al.

Brain Research|December 4, 1992

Corticotropin-releasing factor induces a place aversion independent of its neuroendocrine roleM Cador, S H Ahmed, G F Koob, et al.

European Journal of Pharmacology|December 3, 1985

A novel long-lasting dopamine receptor blocker: haloperidol-bovine serum albumin conjugateA M Garrigues, L Stinus, K Taghzouti, et al.

Psychopharmacology|January 1, 1984

6-Hydroxydopamine lesion of the dopamine mesocorticolimbic cell bodies increases (+)-amphetamine self-administrationJ M Deminière, H Simon, J P Herman, et al.

Neuroscience Letters|July 19, 1988

Intracerebral dopaminergic transplants are not activated by electrical footshock stress activating in situ mesocorticolimbic neuronsJ P Herman, J M Rivet, N Abrous, et al.

Brain Research|June 15, 1984

Neuroleptic-like disruption of the conditioned avoidance response requires destruction of both the mesolimbic and nigrostriatal dopamine systemsG F Koob, H Simon, J P Herman, et al.

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Showing results (31-40 of 471) with videos related to

Sort By:

Pageof 48

Brain Research|June 18, 1984

Functional recovery following transplantation of ventral mesencephalic cells in rat subjected to 6-OHDA lesions of the mesolimbic dopaminergic neuronsD Nadaud, J P Herman, H Simon, et al.

Physiology & Behavior|December 1, 1984

Contributions of the mesencephalic dopaminergic system and the trigeminal sensory pathway to the ventral tegmental aphagia syndrome in ratsD Nadaud, H Simon, J P Herman, et al.

Proceedings of the National Academy of Sciences of the United States of America|April 1, 1980

Locomotor activation induced by infusion of endorphins into the ventral tegmental area: evidence for opiate-dopamine interactionsL Stinus, G F Koob, N Ling, et al.

Brain Research|May 8, 1989

Substance P, neurotensin and enkephalin injections into the ventral tegmental area: comparative study on dopamine turnover in several forebrain structuresM Cador, J M Rivet, A E Kelley, et al.

Brain Research|March 26, 1993

Central administration of corticotropin releasing factor induces long-term sensitization to D-amphetamineM Cador, B J Cole, G F Koob, et al.

Brain Research|December 4, 1992

Corticotropin-releasing factor induces a place aversion independent of its neuroendocrine roleM Cador, S H Ahmed, G F Koob, et al.

European Journal of Pharmacology|December 3, 1985

A novel long-lasting dopamine receptor blocker: haloperidol-bovine serum albumin conjugateA M Garrigues, L Stinus, K Taghzouti, et al.

Psychopharmacology|January 1, 1984

6-Hydroxydopamine lesion of the dopamine mesocorticolimbic cell bodies increases (+)-amphetamine self-administrationJ M Deminière, H Simon, J P Herman, et al.

Neuroscience Letters|July 19, 1988

Intracerebral dopaminergic transplants are not activated by electrical footshock stress activating in situ mesocorticolimbic neuronsJ P Herman, J M Rivet, N Abrous, et al.

Brain Research|June 15, 1984

Neuroleptic-like disruption of the conditioned avoidance response requires destruction of both the mesolimbic and nigrostriatal dopamine systemsG F Koob, H Simon, J P Herman, et al.

Pageof 48