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J Parma

Showing results (11-20 of 69) with videos related to

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The Journal of Clinical Endocrinology and Metabolism|May 20, 1998
Apparent congenital athyreosis contrasting with normal plasma thyroglobulin levels and associated with inactivating mutations in the thyrotropin receptor gene: are athyreosis and ectopic thyroid distinct entities?N Gagné, J Parma, C Deal, et al.
Archives of Environmental Contamination and Toxicology|February 16, 2008
Accumulation and elimination of chromium by freshwater species exposed to spiked sedimentsMercedes Marchese, Ana M Gagneten, María J Parma, et al.
The Journal of Clinical Endocrinology and Metabolism|December 1, 1994
Identification and functional characterization of two new somatic mutations causing constitutive activation of the thyrotropin receptor in hyperfunctioning autonomous adenomas of the thyroidR Paschke, M Tonacchera, J Van Sande, et al.
The Journal of Clinical Endocrinology and Metabolism|April 13, 1999
A germline mutation of the thyrotropin receptor gene associated with thyrotoxicosis and mitral valve prolapse in a Chinese familyD H Khoo, J Parma, C Rajasoorya, et al.
Bulletin of Environmental Contamination and Toxicology|February 19, 2013
Hepatic enzymes activity in the fish Prochilodus lineatus (Valenciennes, 1836) after sublethal cypermethrin exposureA Loteste, J Scagnetti, M F Simoniello, et al.
Thyroid : Official Journal of the American Thyroid Association|November 22, 2001
A familial case of congenital hypothyroidism caused by a homozygous mutation of the thyrotropin receptor geneP Bretones, L Duprez, J Parma, et al.
Acta Clinica Belgica|June 3, 2011
A woman with recurrent "infections" since birth--a new mevalonate kinase mutationC M Farber, J A W Wanders, J C Goffard, et al.
Molecular Endocrinology (Baltimore, Md.)|June 1, 1995
Somatic mutations causing constitutive activity of the thyrotropin receptor are the major cause of hyperfunctioning thyroid adenomas: identification of additional mutations activating both the cyclic adenosine 3',5'-monophosphate and inositol phosphate-Ca2+ cascadesJ Parma, J Van Sande, S Swillens, et al.
Drug and Chemical Toxicology|September 8, 2018
Stress oxidative and genotoxicity in <i>Prochilodus lineatus</i> (Valenciennes, 1836) exposed to commercial formulation of insecticide cypermethrinC E Davico, A Loteste, M J Parma, et al.
Biochemical and Biophysical Research Communications|August 30, 1991
Sequence of a human brain adenylyl cyclase partial cDNA: evidence for a consensus cyclase specific domainJ Parma, D Stengel, M H Gannage, et al.
Pageof 7

Showing results (11-20 of 69) with videos related to

Sort By:
Pageof 7
The Journal of Clinical Endocrinology and Metabolism|May 20, 1998
Apparent congenital athyreosis contrasting with normal plasma thyroglobulin levels and associated with inactivating mutations in the thyrotropin receptor gene: are athyreosis and ectopic thyroid distinct entities?N Gagné, J Parma, C Deal, et al.
Archives of Environmental Contamination and Toxicology|February 16, 2008
Accumulation and elimination of chromium by freshwater species exposed to spiked sedimentsMercedes Marchese, Ana M Gagneten, María J Parma, et al.
The Journal of Clinical Endocrinology and Metabolism|December 1, 1994
Identification and functional characterization of two new somatic mutations causing constitutive activation of the thyrotropin receptor in hyperfunctioning autonomous adenomas of the thyroidR Paschke, M Tonacchera, J Van Sande, et al.
The Journal of Clinical Endocrinology and Metabolism|April 13, 1999
A germline mutation of the thyrotropin receptor gene associated with thyrotoxicosis and mitral valve prolapse in a Chinese familyD H Khoo, J Parma, C Rajasoorya, et al.
Bulletin of Environmental Contamination and Toxicology|February 19, 2013
Hepatic enzymes activity in the fish Prochilodus lineatus (Valenciennes, 1836) after sublethal cypermethrin exposureA Loteste, J Scagnetti, M F Simoniello, et al.
Thyroid : Official Journal of the American Thyroid Association|November 22, 2001
A familial case of congenital hypothyroidism caused by a homozygous mutation of the thyrotropin receptor geneP Bretones, L Duprez, J Parma, et al.
Acta Clinica Belgica|June 3, 2011
A woman with recurrent "infections" since birth--a new mevalonate kinase mutationC M Farber, J A W Wanders, J C Goffard, et al.
Molecular Endocrinology (Baltimore, Md.)|June 1, 1995
Somatic mutations causing constitutive activity of the thyrotropin receptor are the major cause of hyperfunctioning thyroid adenomas: identification of additional mutations activating both the cyclic adenosine 3',5'-monophosphate and inositol phosphate-Ca2+ cascadesJ Parma, J Van Sande, S Swillens, et al.
Drug and Chemical Toxicology|September 8, 2018
Stress oxidative and genotoxicity in <i>Prochilodus lineatus</i> (Valenciennes, 1836) exposed to commercial formulation of insecticide cypermethrinC E Davico, A Loteste, M J Parma, et al.
Biochemical and Biophysical Research Communications|August 30, 1991
Sequence of a human brain adenylyl cyclase partial cDNA: evidence for a consensus cyclase specific domainJ Parma, D Stengel, M H Gannage, et al.
Pageof 7