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Hospital Practice (Office Ed.)|April 15, 1989
Metabolic disturbances after coronary occlusionJ R NeelyCirculation Research|May 1, 1988
Hearts from diabetic rats are more resistant to in vitro ischemia: possible role of altered Ca2+ metabolismM Tani, J R NeelyThe American Journal of Physiology|April 1, 1990
Mechanisms of reduced reperfusion injury by low Ca2+ and/or high K+M Tani, J R NeelyJournal of Molecular and Cellular Cardiology|March 1, 1989
Inhibition of post-ischemic ventricular recovery by low concentrations of prostacyclin in isolated working rat hearts: dependency on concentration, ischemia duration, calcium and relationship to myocardial energy metabolismM Karmazyn, J R NeelyJournal of Molecular and Cellular Cardiology|June 1, 1983
Effects of increased cardiac work on pyruvate dehydrogenase activity in hearts from diabetic animalsK Kobayashi, J R NeelyJournal of Molecular and Cellular Cardiology|June 1, 1983
Effects of ischemia and reperfusion on pyruvate dehydrogenase activity in isolated rat heartsK Kobayashi, J R NeelyJournal of Molecular and Cellular Cardiology|June 1, 1983
Mechanism of pyruvate dehydrogenase activation by increased cardiac workK Kobayashi, J R NeelyCirculation Research|October 1, 1989
Role of intracellular Na+ in Ca2+ overload and depressed recovery of ventricular function of reperfused ischemic rat hearts. Possible involvement of H+-Na+ and Na+-Ca2+ exchangeM Tani, J R NeelyBasic Research in Cardiology|July 1, 1991
Deleterious effects of digitalis on reperfusion-induced arrhythmias and myocardial injury in ischemic rat hearts: possible involvements of myocardial Na+ and Ca2+ imbalanceM Tani, J R NeelyThe American Journal of Pathology|February 1, 1981
Metabolic products and myocardial ischemiaJ R Neely, D FeuvrayPageof 7