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Transfusion|March 10, 2001
The low-incidence MNS antigens M(v), s(D), and Mit arise from single amino acid substitutions on GPBJ R Storry, M E Reid, S MacLennan, et al.Immunohematology|January 1, 1996
Glycophorin A-deficient red cells may have a weak expression of C4-bound Ch and Rg antigensP Tippett, J R Storry, P S Walker, et al.Transfusion|August 1, 1996
Expression and quantitative variation of the low-incidence blood group antigen He on some S-s-red cellsM E Reid, J R Storry, H Ralph, et al.Immunohematology|September 18, 2004
Moderate hemolytic disease of the newborn (HDN) due to anti-Rh17 produced by a black female with an e variant phenotypeM C Brumit, G E Carnahan, J R Stubbs, et al.Vox Sanguinis|December 9, 2000
Four examples of anti-TSEN and three of TSEN-positive erythrocytesJ R Storry, G Lindsay, S Rolih, et al.Transfusion|March 30, 2000
Molecular mechanisms that lead to reduced expression of duffy antigensK Yazdanbakhsh, M Rios, J R Storry, et al.Transfusion|November 29, 2001
Molecular basis of the Dombrock null phenotypeM Rios, K Hue-Roye, J R Storry, et al.Vox Sanguinis|December 9, 2000
First example of hemolytic disease of the newborn caused by anti-Or and confirmation of the molecular basis of OrM E Reid, L Sausais, R Oyen, et al.Transfusion|January 27, 1999
NOR polyagglutination and Sta glycophorin in one family: relation of NOR polyagglutination to terminal alpha-galactose residues and abnormal glycolipidsG Kuśnierz-Alejska, M Duk, J R Storry, et al.Pageof 24