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The New England Journal of Medicine
|
November 13, 2014
Inactivating mutations in NPC1L1 and protection from coronary heart disease
, Nathan O Stitziel, Hong-Hee Won, et al.
American Journal of Human Genetics
|
June 11, 2021
Pan-ancestry exome-wide association analyses of COVID-19 outcomes in 586,157 individuals
Jack A Kosmicki, Julie E Horowitz, Nilanjana Banerjee, et al.
Nature Genetics
|
July 14, 2022
Genetic regulation of OAS1 nonsense-mediated decay underlies association with COVID-19 hospitalization in patients of European and African ancestries
A Rouf Banday, Megan L Stanifer, Oscar Florez-Vargas, et al.
Plos One
|
December 14, 2012
Gene-centric meta-analysis of lipid traits in African, East Asian and Hispanic populations
Clara C Elbers, Yiran Guo, Vinicius Tragante, et al.
Medrxiv : the Preprint Server for Health Sciences
|
March 3, 2021
A catalog of associations between rare coding variants and COVID-19 outcomes
J A Kosmicki, J E Horowitz, N Banerjee, et al.
Nature Genetics
|
May 23, 2017
Fifteen new risk loci for coronary artery disease highlight arterial-wall-specific mechanisms
Joanna M M Howson, Wei Zhao, Daniel R Barnes, et al.
Nature Genetics
|
July 3, 2007
Two variants on chromosome 17 confer prostate cancer risk, and the one in TCF2 protects against type 2 diabetes
Julius Gudmundsson, Patrick Sulem, Valgerdur Steinthorsdottir, et al.
Nature Genetics
|
October 13, 2009
A genome-wide meta-analysis identifies 22 loci associated with eight hematological parameters in the HaemGen consortium
Nicole Soranzo, Tim D Spector, Massimo Mangino, et al.
Circulation. Cardiovascular Genetics
|
May 25, 2011
A genome-wide association study identifies LIPA as a susceptibility gene for coronary artery disease
Philipp S Wild, Tanja Zeller, Arne Schillert, et al.
Nature Communications
|
June 15, 2018
Genetic inactivation of ANGPTL4 improves glucose homeostasis and is associated with reduced risk of diabetes
Viktoria Gusarova, Colm O'Dushlaine, Tanya M Teslovich, et al.
Page
of 90
Search research articles
Search
Showing results (841-850 of 897) with videos related to
Sort By:
Page
of 90
The New England Journal of Medicine
|
November 13, 2014
Inactivating mutations in NPC1L1 and protection from coronary heart disease
, Nathan O Stitziel, Hong-Hee Won, et al.
American Journal of Human Genetics
|
June 11, 2021
Pan-ancestry exome-wide association analyses of COVID-19 outcomes in 586,157 individuals
Jack A Kosmicki, Julie E Horowitz, Nilanjana Banerjee, et al.
Nature Genetics
|
July 14, 2022
Genetic regulation of OAS1 nonsense-mediated decay underlies association with COVID-19 hospitalization in patients of European and African ancestries
A Rouf Banday, Megan L Stanifer, Oscar Florez-Vargas, et al.
Plos One
|
December 14, 2012
Gene-centric meta-analysis of lipid traits in African, East Asian and Hispanic populations
Clara C Elbers, Yiran Guo, Vinicius Tragante, et al.
Medrxiv : the Preprint Server for Health Sciences
|
March 3, 2021
A catalog of associations between rare coding variants and COVID-19 outcomes
J A Kosmicki, J E Horowitz, N Banerjee, et al.
Nature Genetics
|
May 23, 2017
Fifteen new risk loci for coronary artery disease highlight arterial-wall-specific mechanisms
Joanna M M Howson, Wei Zhao, Daniel R Barnes, et al.
Nature Genetics
|
July 3, 2007
Two variants on chromosome 17 confer prostate cancer risk, and the one in TCF2 protects against type 2 diabetes
Julius Gudmundsson, Patrick Sulem, Valgerdur Steinthorsdottir, et al.
Nature Genetics
|
October 13, 2009
A genome-wide meta-analysis identifies 22 loci associated with eight hematological parameters in the HaemGen consortium
Nicole Soranzo, Tim D Spector, Massimo Mangino, et al.
Circulation. Cardiovascular Genetics
|
May 25, 2011
A genome-wide association study identifies LIPA as a susceptibility gene for coronary artery disease
Philipp S Wild, Tanja Zeller, Arne Schillert, et al.
Nature Communications
|
June 15, 2018
Genetic inactivation of ANGPTL4 improves glucose homeostasis and is associated with reduced risk of diabetes
Viktoria Gusarova, Colm O'Dushlaine, Tanya M Teslovich, et al.
Page
of 90