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The Biochemical Journal|December 1, 1993
Involvement of human plasma angiotensin I-converting enzyme in the degradation of the haemoregulatory peptide N-acetyl-seryl-aspartyl-lysyl-prolineK J Rieger, N Saez-Servent, M P Papet, et al.International Journal of Cancer|April 30, 2025
Tyrosine kinase inhibitors with blinatumomab versus chemotherapy in Philadelphia-positive acute B-lymphoblastic leukemiaSebastian M Stolz, Kevin D Hofer, Wiebke Rösler, et al.Journal of Medical Genetics|April 1, 1994
Linkage analysis of families with severe childhood autosomal recessive muscular dystrophy in Morocco indicates genetic homogeneity of the disease in north AfricaF el Kerch, A Sefiani, K Azibi, et al.Genomics|July 15, 1994
Refined mapping of a gene (NPH1) causing familial juvenile nephronophthisis and evidence for genetic heterogeneityM Medhioub, D Cherif, F Benessy, et al.Journal of Neurology|September 29, 2005
Adult medulloblastoma: prognostic factors and response to therapy at diagnosis and at relapseUlrich Herrlinger, A Steinbrecher, J Rieger, et al.International Journal of Cancer|December 8, 1998
Predicting chemoresistance in human malignant glioma cells: the role of molecular genetic analysesM Weller, J Rieger, C Grimmel, et al.Cell Death and Differentiation|October 29, 2005
BCL-xL: time-dependent dissociation between modulation of apoptosis and invasiveness in human malignant glioma cellsM Weiler, O Bähr, U Hohlweg, et al.American Journal of Human Genetics|December 1, 1992
Study of large inbred Friedreich ataxia families reveals a recombination between D9S15 and the disease locusS Belal, K Panayides, G Sirugo, et al.Neurology|November 1, 1993
Friedreich's ataxia phenotype not linked to chromosome 9 and associated with selective autosomal recessive vitamin E deficiency in two inbred Tunisian familiesM Ben Hamida, S Belal, G Sirugo, et al.Plos One|October 12, 2013
Indoor navigation by people with visual impairment using a digital sign systemGordon E Legge, Paul J Beckmann, Bosco S Tjan, et al.Pageof 45