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J SACHS

Showing results (201-210 of 282) with videos related to

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Transfusion|March 5, 2019
The nonconservative CD177 single-nucleotide polymorphism c.1291G>A is a genetic determinant for human neutrophil antigen-2 atypical/low expression and deficiencyJianming Wu, Yunfang Li, Randy M Schuller, et al.
Investigative Ophthalmology & Visual Science|January 1, 2010
The safety of intraocular linezolid in rabbitsSara L Duke, Leila I Kump, Yang Yuan, et al.
Journal of Neurology, Neurosurgery, and Psychiatry|February 1, 1979
HLA antigens in chronic relapsing idiopathic inflammatory polyneuropathyD Adams, H Festenstein, J D Gibson, et al.
Bulletin of the World Health Organization|September 8, 2001
Round table. A role for public-private partnerships in controlling neglected diseases?R G Ridley, J Lob-Levyt, J Sachs, et al.
Lancet (London, England)|September 3, 1977
HLA antigens in Guillain-Barré syndromeD Adams, J D Gibson, P K Thomas, et al.
Journal of the American College of Surgeons|September 14, 2022
NSQIP Quality Benchmarking and Evaluation of Potential Bias Associated with Higher- or Lower-Risk Operation Case MixMark E Cohen, Yaoming Liu, Bruce L Hall, et al.
British Journal of Haematology|January 31, 2020
Maternal antibodies against paternal class I human leukocyte antigens are not associated with foetal and neonatal alloimmune thrombocytopeniaUlrich J Sachs, Sandra Wienzek-Lischka, Yalin Duong, et al.
The Journal of Biological Chemistry|October 16, 2004
The junctional adhesion molecule-C promotes neutrophil transendothelial migration in vitro and in vivoTriantafyllos Chavakis, Tanja Keiper, Rachel Matz-Westphal, et al.
Blood|May 7, 2013
Inhibition of HPA-1a alloantibody-mediated platelet destruction by a deglycosylated anti-HPA-1a monoclonal antibody in mice: toward targeted treatment of fetal-alloimmune thrombocytopeniaTamam Bakchoul, Andreas Greinacher, Ulrich J Sachs, et al.
Thrombosis and Haemostasis|November 26, 2011
A point mutation in the EGF-4 domain of β(3) integrin is responsible for the formation of the Sec(a) platelet alloantigen and affects receptor functionUlrich J Sachs, Tamam Bakchoul, Olga Eva, et al.
Pageof 29

Showing results (201-210 of 282) with videos related to

Sort By:
Pageof 29
Transfusion|March 5, 2019
The nonconservative CD177 single-nucleotide polymorphism c.1291G>A is a genetic determinant for human neutrophil antigen-2 atypical/low expression and deficiencyJianming Wu, Yunfang Li, Randy M Schuller, et al.
Investigative Ophthalmology & Visual Science|January 1, 2010
The safety of intraocular linezolid in rabbitsSara L Duke, Leila I Kump, Yang Yuan, et al.
Journal of Neurology, Neurosurgery, and Psychiatry|February 1, 1979
HLA antigens in chronic relapsing idiopathic inflammatory polyneuropathyD Adams, H Festenstein, J D Gibson, et al.
Bulletin of the World Health Organization|September 8, 2001
Round table. A role for public-private partnerships in controlling neglected diseases?R G Ridley, J Lob-Levyt, J Sachs, et al.
Lancet (London, England)|September 3, 1977
HLA antigens in Guillain-Barré syndromeD Adams, J D Gibson, P K Thomas, et al.
Journal of the American College of Surgeons|September 14, 2022
NSQIP Quality Benchmarking and Evaluation of Potential Bias Associated with Higher- or Lower-Risk Operation Case MixMark E Cohen, Yaoming Liu, Bruce L Hall, et al.
British Journal of Haematology|January 31, 2020
Maternal antibodies against paternal class I human leukocyte antigens are not associated with foetal and neonatal alloimmune thrombocytopeniaUlrich J Sachs, Sandra Wienzek-Lischka, Yalin Duong, et al.
The Journal of Biological Chemistry|October 16, 2004
The junctional adhesion molecule-C promotes neutrophil transendothelial migration in vitro and in vivoTriantafyllos Chavakis, Tanja Keiper, Rachel Matz-Westphal, et al.
Blood|May 7, 2013
Inhibition of HPA-1a alloantibody-mediated platelet destruction by a deglycosylated anti-HPA-1a monoclonal antibody in mice: toward targeted treatment of fetal-alloimmune thrombocytopeniaTamam Bakchoul, Andreas Greinacher, Ulrich J Sachs, et al.
Thrombosis and Haemostasis|November 26, 2011
A point mutation in the EGF-4 domain of β(3) integrin is responsible for the formation of the Sec(a) platelet alloantigen and affects receptor functionUlrich J Sachs, Tamam Bakchoul, Olga Eva, et al.
Pageof 29