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J Sparkowski

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Genetics|March 1, 1992
Simultaneous gain and loss of functions caused by a single amino acid substitution in the beta subunit of Escherichia coli RNA polymerase: suppression of nusA and rho mutations and conditional lethalityJ Sparkowski, A Das
The EMBO Journal|July 3, 1995
E5 oncoprotein retained in the endoplasmic reticulum/cis Golgi still induces PDGF receptor autophosphorylation but does not transform cellsJ Sparkowski, J Anders, R Schlegel
Journal of Virology|September 1, 1994
Mutation of the bovine papillomavirus E5 oncoprotein at amino acid 17 generates both high- and low-transforming variantsJ Sparkowski, J Anders, R Schlegel
Journal of Virology|April 1, 1996
E5 oncoprotein transmembrane mutants dissociate fibroblast transforming activity from 16-kilodalton protein binding and platelet-derived growth factor receptor binding and phosphorylationJ Sparkowski, M Mense, J Anders, et al.
The Journal of Biological Chemistry|February 15, 2000
E5 oncoprotein mutants activate phosphoinositide 3-kinase independently of platelet-derived growth factor receptor activationF A Suprynowicz, J Sparkowski, A Baege, et al.
The Journal of Biological Chemistry|March 24, 1995
Vacuolar H(+)-ATPase mutants transform cells and define a binding site for the papillomavirus E5 oncoproteinT Andresson, J Sparkowski, D J Goldstein, et al.
The EMBO Journal|December 1, 1992
The BPV-1 E5 protein, the 16 kDa membrane pore-forming protein and the PDGF receptor exist in a complex that is dependent on hydrophobic transmembrane interactionsD J Goldstein, T Andresson, J J Sparkowski, et al.
Virology|April 2, 2003
Expression of the bovine papillomavirus type 1 E5B gene reveals a protein-protein interaction of the E5A and E5B gene productsA P Wlazlo, J J Sparkowski, A B Jenson, et al.
The Journal of Cell Biology|January 29, 2000
Golgi alkalinization by the papillomavirus E5 oncoproteinF Schapiro, J Sparkowski, A Adduci, et al.
Gynecologic Oncology|May 1, 1997
Identification of H, K, and N-ras point mutations in stage IB cervical carcinomaE C Grendys, W A Barnes, J Weitzel, et al.
Pageof 2

Showing results (1-10 of 14) with videos related to

Sort By:
Pageof 2
Genetics|March 1, 1992
Simultaneous gain and loss of functions caused by a single amino acid substitution in the beta subunit of Escherichia coli RNA polymerase: suppression of nusA and rho mutations and conditional lethalityJ Sparkowski, A Das
The EMBO Journal|July 3, 1995
E5 oncoprotein retained in the endoplasmic reticulum/cis Golgi still induces PDGF receptor autophosphorylation but does not transform cellsJ Sparkowski, J Anders, R Schlegel
Journal of Virology|September 1, 1994
Mutation of the bovine papillomavirus E5 oncoprotein at amino acid 17 generates both high- and low-transforming variantsJ Sparkowski, J Anders, R Schlegel
Journal of Virology|April 1, 1996
E5 oncoprotein transmembrane mutants dissociate fibroblast transforming activity from 16-kilodalton protein binding and platelet-derived growth factor receptor binding and phosphorylationJ Sparkowski, M Mense, J Anders, et al.
The Journal of Biological Chemistry|February 15, 2000
E5 oncoprotein mutants activate phosphoinositide 3-kinase independently of platelet-derived growth factor receptor activationF A Suprynowicz, J Sparkowski, A Baege, et al.
The Journal of Biological Chemistry|March 24, 1995
Vacuolar H(+)-ATPase mutants transform cells and define a binding site for the papillomavirus E5 oncoproteinT Andresson, J Sparkowski, D J Goldstein, et al.
The EMBO Journal|December 1, 1992
The BPV-1 E5 protein, the 16 kDa membrane pore-forming protein and the PDGF receptor exist in a complex that is dependent on hydrophobic transmembrane interactionsD J Goldstein, T Andresson, J J Sparkowski, et al.
Virology|April 2, 2003
Expression of the bovine papillomavirus type 1 E5B gene reveals a protein-protein interaction of the E5A and E5B gene productsA P Wlazlo, J J Sparkowski, A B Jenson, et al.
The Journal of Cell Biology|January 29, 2000
Golgi alkalinization by the papillomavirus E5 oncoproteinF Schapiro, J Sparkowski, A Adduci, et al.
Gynecologic Oncology|May 1, 1997
Identification of H, K, and N-ras point mutations in stage IB cervical carcinomaE C Grendys, W A Barnes, J Weitzel, et al.
Pageof 2