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J Sturis

Showing results (51-60 of 60) with videos related to

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Diabetes, Obesity & Metabolism|July 2, 2010
Insulin detemir is a fully efficacious, low affinity agonist at the insulin receptorA R Sørensen, C E Stidsen, U Ribel, et al.
The Journal of Clinical Investigation|March 1, 1994
Insulin secretory abnormalities in subjects with hyperglycemia due to glucokinase mutationsM M Byrne, J Sturis, K Clément, et al.
Diabetes|June 1, 1993
Oscillatory insulin secretion after pancreas transplantN M O'Meara, J Sturis, J D Blackman, et al.
Diabetes|January 1, 1994
Abnormal insulin secretion, not insulin resistance, is the genetic or primary defect of MODY in the RW pedigreeW H Herman, S S Fajans, F J Ortiz, et al.
The American Journal of Physiology|January 11, 1996
NIDDM is associated with loss of pancreatic beta-cell L-type Ca2+ channel activityM W Roe, J F Worley, Y Tokuyama, et al.
The American Journal of Physiology|June 1, 1994
Demonstration of rapid light-induced advances and delays of the human circadian clock using hormonal phase markersE Van Cauter, J Sturis, M M Byrne, et al.
Diabetes|June 1, 1995
Altered insulin secretory responses to glucose in subjects with a mutation in the MODY1 gene on chromosome 20M M Byrne, J Sturis, S S Fajans, et al.
The American Journal of Physiology|June 1, 1994
Nocturnal exercise phase delays circadian rhythms of melatonin and thyrotropin secretion in normal menO Van Reeth, J Sturis, M M Byrne, et al.
Diabetes|April 1, 1996
Clinical phenotypes, insulin secretion, and insulin sensitivity in kindreds with maternally inherited diabetes and deafness due to mitochondrial tRNALeu(UUR) gene mutationG Velho, M M Byrne, K Clément, et al.
Diabetes|November 1, 1996
Altered insulin secretory responses to glucose in diabetic and nondiabetic subjects with mutations in the diabetes susceptibility gene MODY3 on chromosome 12M M Byrne, J Sturis, S Menzel, et al.
Pageof 6

Showing results (51-60 of 60) with videos related to

Sort By:
Pageof 6
You have reached the last page of results.This site can display upto 60 results.
Diabetes, Obesity & Metabolism|July 2, 2010
Insulin detemir is a fully efficacious, low affinity agonist at the insulin receptorA R Sørensen, C E Stidsen, U Ribel, et al.
The Journal of Clinical Investigation|March 1, 1994
Insulin secretory abnormalities in subjects with hyperglycemia due to glucokinase mutationsM M Byrne, J Sturis, K Clément, et al.
Diabetes|June 1, 1993
Oscillatory insulin secretion after pancreas transplantN M O'Meara, J Sturis, J D Blackman, et al.
Diabetes|January 1, 1994
Abnormal insulin secretion, not insulin resistance, is the genetic or primary defect of MODY in the RW pedigreeW H Herman, S S Fajans, F J Ortiz, et al.
The American Journal of Physiology|January 11, 1996
NIDDM is associated with loss of pancreatic beta-cell L-type Ca2+ channel activityM W Roe, J F Worley, Y Tokuyama, et al.
The American Journal of Physiology|June 1, 1994
Demonstration of rapid light-induced advances and delays of the human circadian clock using hormonal phase markersE Van Cauter, J Sturis, M M Byrne, et al.
Diabetes|June 1, 1995
Altered insulin secretory responses to glucose in subjects with a mutation in the MODY1 gene on chromosome 20M M Byrne, J Sturis, S S Fajans, et al.
The American Journal of Physiology|June 1, 1994
Nocturnal exercise phase delays circadian rhythms of melatonin and thyrotropin secretion in normal menO Van Reeth, J Sturis, M M Byrne, et al.
Diabetes|April 1, 1996
Clinical phenotypes, insulin secretion, and insulin sensitivity in kindreds with maternally inherited diabetes and deafness due to mitochondrial tRNALeu(UUR) gene mutationG Velho, M M Byrne, K Clément, et al.
Diabetes|November 1, 1996
Altered insulin secretory responses to glucose in diabetic and nondiabetic subjects with mutations in the diabetes susceptibility gene MODY3 on chromosome 12M M Byrne, J Sturis, S Menzel, et al.
Pageof 6