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Brain Research|October 21, 1985
Basal forebrain lesions produce a dissociation of trial-dependent and trial-independent memory performanceB J Knowlton, G L Wenk, D S Olton, et al.Journal of Neurochemistry|October 1, 1986
Synaptosomal transport of radiolabel from N-acetyl-aspartyl-[3H]glutamate suggests a mechanism of inactivation of an excitatory neuropeptideR D Blakely, L Ory-Lavollée, R C Thompson, et al.Brain Research|November 4, 1985
Regional brain levels of N-acetyl-aspartyl-glutamate: the effect of kindled seizuresJ L Meyerhoff, K J Koller, D D Walczak, et al.Journal of Neurochemistry|September 15, 2000
Insulin-like growth factor I prevents the development of sensitivity to kainate neurotoxicity in cerebellar granule cellsM L Leski, S L Valentine, J D Baer, et al.Behavioral Neuroscience|February 1, 1984
Cholinergic lesion of the striatum impairs acquisition and retention of a passive avoidance responseK Sandberg, P R Sanberg, I Hanin, et al.European Journal of Pharmacology|July 14, 1988
A re-examination of the interaction of N-acetyl-L-aspartyl-L-glutamate with a subpopulation of rat brain membrane L-[3H]glutamate binding sitesR D Blakely, M B Robinson, A S Guarda, et al.Brain Research|August 1, 1984
Morphologic and neurochemical studies of embryonic brain development in murine trisomy 16H S Singer, M Tiemeyer, J C Hedreen, et al.Epilepsia|November 1, 1986
Dicarboxylic amino acids block epileptiform activity in hippocampal sliceJ M ffrench-Mullen, T H Murphy, J T Coyle, et al.Journal of Neurochemistry|December 1, 1988
Calcium-dependent evoked release of N-[3H]acetylaspartylglutamate from the optic pathwayG Tsai, G Forloni, M B Robinson, et al.Neuropharmacology|April 1, 1987
Characteristics of chloride-dependent incorporation of glutamate into brain membranes argue against a receptor binding siteR Zaczek, S Arlis, A Markl, et al.Pageof 28