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J Tomasek

Showing results (61-70 of 71) with videos related to

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Neuro-Oncology Advances|November 26, 2019
Sox2<sup>+</sup> cells in Sonic Hedgehog-subtype medulloblastoma resist p53-mediated cell-cycle arrest response and drive therapy-induced recurrenceDaniel M Treisman, Yinghua Li, Brianna R Pierce, et al.
Investigative Ophthalmology & Visual Science|May 26, 2006
Deletion of smooth muscle alpha-actin alters blood-retina barrier permeability and retinal functionJames J Tomasek, Carol J Haaksma, Robert J Schwartz, et al.
Experimental Cell Research|March 28, 2012
MMP-2 expression by fibroblasts is suppressed by the myofibroblast phenotypeEric W Howard, Beverly J Crider, Dawn L Updike, et al.
The Journal of Urology|September 17, 2004
Decreased expression of smooth muscle alpha-actin results in decreased contractile function of the mouse bladderR A Zimmerman, J J Tomasek, J McRae, et al.
Neoplasma|July 13, 2011
Clinical significance of the plasminogen activator system in relation to grade of tumor and treatment response in colorectal carcinoma patientsJ Halamkova, I Kiss, Z Pavlovsky, et al.
The Journal of Investigative Dermatology|January 28, 2014
Elevated circulation levels of an antiangiogenic SERPIN in patients with diabetic microvascular complications impair wound healing through suppression of Wnt signalingJeffrey D McBride, Alicia J Jenkins, Xiaochen Liu, et al.
Neoplasma|December 25, 2012
Clinical impact of PAI 1 4G/5G gene polymorphism in colorectal carcinoma patientsJ Halamkova, I Kiss, Z Pavlovsky, et al.
Cancer Research|June 12, 2016
Survival of Patients with Gastrointestinal Cancers Can Be Predicted by a Surrogate microRNA Signature for Cancer Stem-like Cells Marked by DCLK1 KinaseNathaniel Weygant, Yang Ge, Dongfeng Qu, et al.
International Journal of Cancer|March 27, 2018
Alternative splice variants of DCLK1 mark cancer stem cells, promote self-renewal and drug-resistance, and can be targeted to inhibit tumorigenesis in kidney cancerYang Ge, Nathaniel Weygant, Dongfeng Qu, et al.
Oncotarget|January 22, 2015
DCLK1 is a broadly dysregulated target against epithelial-mesenchymal transition, focal adhesion, and stemness in clear cell renal carcinomaNathaniel Weygant, Dongfeng Qu, Randal May, et al.
Pageof 8

Showing results (61-70 of 71) with videos related to

Sort By:
Pageof 8
Neuro-Oncology Advances|November 26, 2019
Sox2<sup>+</sup> cells in Sonic Hedgehog-subtype medulloblastoma resist p53-mediated cell-cycle arrest response and drive therapy-induced recurrenceDaniel M Treisman, Yinghua Li, Brianna R Pierce, et al.
Investigative Ophthalmology & Visual Science|May 26, 2006
Deletion of smooth muscle alpha-actin alters blood-retina barrier permeability and retinal functionJames J Tomasek, Carol J Haaksma, Robert J Schwartz, et al.
Experimental Cell Research|March 28, 2012
MMP-2 expression by fibroblasts is suppressed by the myofibroblast phenotypeEric W Howard, Beverly J Crider, Dawn L Updike, et al.
The Journal of Urology|September 17, 2004
Decreased expression of smooth muscle alpha-actin results in decreased contractile function of the mouse bladderR A Zimmerman, J J Tomasek, J McRae, et al.
Neoplasma|July 13, 2011
Clinical significance of the plasminogen activator system in relation to grade of tumor and treatment response in colorectal carcinoma patientsJ Halamkova, I Kiss, Z Pavlovsky, et al.
The Journal of Investigative Dermatology|January 28, 2014
Elevated circulation levels of an antiangiogenic SERPIN in patients with diabetic microvascular complications impair wound healing through suppression of Wnt signalingJeffrey D McBride, Alicia J Jenkins, Xiaochen Liu, et al.
Neoplasma|December 25, 2012
Clinical impact of PAI 1 4G/5G gene polymorphism in colorectal carcinoma patientsJ Halamkova, I Kiss, Z Pavlovsky, et al.
Cancer Research|June 12, 2016
Survival of Patients with Gastrointestinal Cancers Can Be Predicted by a Surrogate microRNA Signature for Cancer Stem-like Cells Marked by DCLK1 KinaseNathaniel Weygant, Yang Ge, Dongfeng Qu, et al.
International Journal of Cancer|March 27, 2018
Alternative splice variants of DCLK1 mark cancer stem cells, promote self-renewal and drug-resistance, and can be targeted to inhibit tumorigenesis in kidney cancerYang Ge, Nathaniel Weygant, Dongfeng Qu, et al.
Oncotarget|January 22, 2015
DCLK1 is a broadly dysregulated target against epithelial-mesenchymal transition, focal adhesion, and stemness in clear cell renal carcinomaNathaniel Weygant, Dongfeng Qu, Randal May, et al.
Pageof 8