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Neuron|December 7, 2002
Synaptotagmin I functions as a calcium sensor to synchronize neurotransmitter releaseMotojiro Yoshihara, J Troy LittletonCurrent Trends in Neurology|December 20, 2011
The biological function of the Huntingtin protein and its relevance to Huntington's Disease pathologyJoost Schulte, J Troy LittletonThe Journal of Neuroscience : the Official Journal of the Society for Neuroscience|February 26, 2017
Pathogenic Huntington Alters BMP Signaling and Synaptic Growth through Local Disruptions of Endosomal CompartmentsYulia Akbergenova, J Troy LittletonFly|September 12, 2013
Modeling Huntington disease in Drosophila: Insights into axonal transport defects and modifiers of toxicityMegan Krench, J Troy LittletonProceedings of the National Academy of Sciences of the United States of America|March 17, 2015
Transmembrane tethering of synaptotagmin to synaptic vesicles controls multiple modes of neurotransmitter releaseJihye Lee, J Troy LittletonNature Neuroscience|September 18, 2007
A complexin fusion clamp regulates spontaneous neurotransmitter release and synaptic growthSarah Huntwork, J Troy LittletonCurrent Biology : CB|May 24, 2011
Synaptic growth: dancing with adducinRobin J Stevens, J Troy LittletonThe Journal of Neuroscience : the Official Journal of the Society for Neuroscience|January 18, 2013
Mutation of a NCKX eliminates glial microdomain calcium oscillations and enhances seizure susceptibilityJan E Melom, J Troy LittletonJournal of Neurogenetics|June 17, 2016
Characterization of axonal transport defects in Drosophila Huntingtin mutantsKurt R Weiss, J Troy LittletonPageof 25