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Oncogene|September 20, 2005
Selective destruction of glioblastoma cells by interference with the activity or expression of ATF5J M Angelastro, P D Canoll, J Kuo, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|July 5, 2001
Death in the balance: alternative participation of the caspase-2 and -9 pathways in neuronal death induced by nerve growth factor deprivationC M Troy, S A Rabacchi, J B Hohl, et al.
The Journal of Biological Chemistry|March 30, 2001
Characterization of a novel isoform of caspase-9 that inhibits apoptosisJ M Angelastro, N Y Moon, D X Liu, et al.
Journal of Combinatorial Chemistry|July 13, 2000
Structure-based design and solid-phase parallel synthesis of phosphorylated nonpeptides to explore hydrophobic binding at the Src SH2 domainC A Metcalf, C J Eyermann, R S Bohacek, et al.
Proceedings of the National Academy of Sciences of the United States of America|December 10, 1999
Mammalian Trithorax and polycomb-group homologues are antagonistic regulators of homeotic developmentR D Hanson, J L Hess, B D Yu, et al.
Chemistry & Biology|March 14, 2000
A Src SH2 selective binding compound inhibits osteoclast-mediated resorptionS M Violette, W C Shakespeare, C Bartlett, et al.
The Journal of Biological Chemistry|November 29, 1996
Induction of CPP32-like activity in PC12 cells by withdrawal of trophic support. Dissociation from apoptosisL Stefanis, D S Park, C Y Yan, et al.
Journal of Neurochemistry|March 30, 2001
beta-Amyloid-induced neuronal apoptosis requires c-Jun N-terminal kinase activationC M Troy, S A Rabacchi, Z Xu, et al.
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