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J-F Dufour

Showing results (11-20 of 56) with videos related to

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Hepatology (Baltimore, Md.)|October 1, 1992
Hepatic accumulation of lysosomes and defective transcytotic vesicular pathways in cirrhotic rat liverJ F Dufour, P Gehr, J Reichen
Journal of Hepatology|April 1, 1994
Increased hepatic lysosomal activity in biliary cirrhosis originates from hepatocytes rather than from macrophagesJ F Dufour, A Zimmermann, J Reichen
Zeitschrift Fur Gastroenterologie|October 17, 2003
Spontaneous regression of an inflammatory pseudotumor of the liverE Biecker, A Zimmermann, J-F Dufour
Digestive Diseases and Sciences|January 9, 1999
Regression of hepatic fibrosis in hepatitis C with long-term interferon treatmentJ F Dufour, R DeLellis, M M Kaplan
Annals of Internal Medicine|December 31, 1997
Reversibility of hepatic fibrosis in autoimmune hepatitisJ F Dufour, R DeLellis, M M Kaplan
Hepatology (Baltimore, Md.)|September 25, 1999
Expression of inositol 1,4,5-trisphosphate receptor isoforms in rat cirrhosisJ F Dufour, M Lüthi, M Forestier, et al.
The Journal of Biological Chemistry|January 31, 1997
Inositol 1,4,5-trisphosphate and calcium regulate the calcium channel function of the hepatic inositol 1,4,5-trisphosphate receptorJ F Dufour, I M Arias, T J Turner
European Journal of Biochemistry|November 28, 2001
Rat inositol 1,4,5-trisphosphate receptor isoform 2 interacts with itself in its C-terminal portion and upstream of the first transmembrane domainF Magnino, K Schmidt, L Mery, et al.
Gastroenterology|March 1, 1995
Nitric oxide blocks bile canalicular contraction by inhibiting inositol trisphosphate-dependent calcium mobilizationJ F Dufour, T J Turner, I M Arias
Progress in Biophysics and Molecular Biology|May 15, 2004
A comparison of three models of the inositol trisphosphate receptorJ Sneyd, M Falcke, J-F Dufour, et al.
Pageof 6

Showing results (11-20 of 56) with videos related to

Sort By:
Pageof 6
Hepatology (Baltimore, Md.)|October 1, 1992
Hepatic accumulation of lysosomes and defective transcytotic vesicular pathways in cirrhotic rat liverJ F Dufour, P Gehr, J Reichen
Journal of Hepatology|April 1, 1994
Increased hepatic lysosomal activity in biliary cirrhosis originates from hepatocytes rather than from macrophagesJ F Dufour, A Zimmermann, J Reichen
Zeitschrift Fur Gastroenterologie|October 17, 2003
Spontaneous regression of an inflammatory pseudotumor of the liverE Biecker, A Zimmermann, J-F Dufour
Digestive Diseases and Sciences|January 9, 1999
Regression of hepatic fibrosis in hepatitis C with long-term interferon treatmentJ F Dufour, R DeLellis, M M Kaplan
Annals of Internal Medicine|December 31, 1997
Reversibility of hepatic fibrosis in autoimmune hepatitisJ F Dufour, R DeLellis, M M Kaplan
Hepatology (Baltimore, Md.)|September 25, 1999
Expression of inositol 1,4,5-trisphosphate receptor isoforms in rat cirrhosisJ F Dufour, M Lüthi, M Forestier, et al.
The Journal of Biological Chemistry|January 31, 1997
Inositol 1,4,5-trisphosphate and calcium regulate the calcium channel function of the hepatic inositol 1,4,5-trisphosphate receptorJ F Dufour, I M Arias, T J Turner
European Journal of Biochemistry|November 28, 2001
Rat inositol 1,4,5-trisphosphate receptor isoform 2 interacts with itself in its C-terminal portion and upstream of the first transmembrane domainF Magnino, K Schmidt, L Mery, et al.
Gastroenterology|March 1, 1995
Nitric oxide blocks bile canalicular contraction by inhibiting inositol trisphosphate-dependent calcium mobilizationJ F Dufour, T J Turner, I M Arias
Progress in Biophysics and Molecular Biology|May 15, 2004
A comparison of three models of the inositol trisphosphate receptorJ Sneyd, M Falcke, J-F Dufour, et al.
Pageof 6