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Jaap J Plomp

Showing results (51-60 of 68) with videos related to

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Plos One|December 5, 2013
The effect of PPE-induced emphysema and chronic LPS-induced pulmonary inflammation on atherosclerosis development in APOE*3-LEIDEN miceP Padmini S J Khedoe, Man C Wong, Gerry T M Wagenaar, et al.
Experimental Neurology|March 10, 2019
Efgartigimod improves muscle weakness in a mouse model for muscle-specific kinase myasthenia gravisMaartje G Huijbers, Jaap J Plomp, Inge E van Es, et al.
Neurology(R) Neuroimmunology & Neuroinflammation|March 19, 2019
MuSK myasthenia gravis monoclonal antibodies: Valency dictates pathogenicityMaartje G Huijbers, Dana L Vergoossen, Yvonne E Fillié-Grijpma, et al.
Genesis (New York, N.Y. : 2000)|December 6, 2006
Conditional inactivation of the Cacna1a gene in transgenic miceBoyan Todorov, Rob C G van de Ven, Simon Kaja, et al.
Plos One|September 15, 2017
Acute and chronic effects of treatment with mesenchymal stromal cells on LPS-induced pulmonary inflammation, emphysema and atherosclerosis developmentP Padmini S J Khedoe, Stan de Kleijn, Annemarie M van Oeveren-Rietdijk, et al.
Neuromuscular Disorders : NMD|January 31, 2012
Comparison of skeletal muscle pathology and motor function of dystrophin and utrophin deficient mouse strainsMaaike van Putten, Darshan Kumar, Margriet Hulsker, et al.
Annals of the New York Academy of Sciences|January 23, 2018
Passive transfer models of myasthenia gravis with muscle-specific kinase antibodiesJan J G M Verschuuren, Jaap J Plomp, Steve J Burden, et al.
The European Journal of Neuroscience|April 19, 2007
Severely impaired neuromuscular synaptic transmission causes muscle weakness in the Cacna1a-mutant mouse rolling NagoyaSimon Kaja, Rob C G van de Ven, J Gert van Dijk, et al.
Science Translational Medicine|March 1, 2019
An automated hybrid bioelectronic system for autogenous restoration of sinus rhythm in atrial fibrillationEmile C A Nyns, René H Poelma, Linda Volkers, et al.
Proceedings of the National Academy of Sciences of the United States of America|March 23, 2021
Functional monovalency amplifies the pathogenicity of anti-MuSK IgG4 in myasthenia gravisDana L E Vergoossen, Jaap J Plomp, Christoph Gstöttner, et al.
Pageof 7

Showing results (51-60 of 68) with videos related to

Sort By:
Pageof 7
Plos One|December 5, 2013
The effect of PPE-induced emphysema and chronic LPS-induced pulmonary inflammation on atherosclerosis development in APOE*3-LEIDEN miceP Padmini S J Khedoe, Man C Wong, Gerry T M Wagenaar, et al.
Experimental Neurology|March 10, 2019
Efgartigimod improves muscle weakness in a mouse model for muscle-specific kinase myasthenia gravisMaartje G Huijbers, Jaap J Plomp, Inge E van Es, et al.
Neurology(R) Neuroimmunology & Neuroinflammation|March 19, 2019
MuSK myasthenia gravis monoclonal antibodies: Valency dictates pathogenicityMaartje G Huijbers, Dana L Vergoossen, Yvonne E Fillié-Grijpma, et al.
Genesis (New York, N.Y. : 2000)|December 6, 2006
Conditional inactivation of the Cacna1a gene in transgenic miceBoyan Todorov, Rob C G van de Ven, Simon Kaja, et al.
Plos One|September 15, 2017
Acute and chronic effects of treatment with mesenchymal stromal cells on LPS-induced pulmonary inflammation, emphysema and atherosclerosis developmentP Padmini S J Khedoe, Stan de Kleijn, Annemarie M van Oeveren-Rietdijk, et al.
Neuromuscular Disorders : NMD|January 31, 2012
Comparison of skeletal muscle pathology and motor function of dystrophin and utrophin deficient mouse strainsMaaike van Putten, Darshan Kumar, Margriet Hulsker, et al.
Annals of the New York Academy of Sciences|January 23, 2018
Passive transfer models of myasthenia gravis with muscle-specific kinase antibodiesJan J G M Verschuuren, Jaap J Plomp, Steve J Burden, et al.
The European Journal of Neuroscience|April 19, 2007
Severely impaired neuromuscular synaptic transmission causes muscle weakness in the Cacna1a-mutant mouse rolling NagoyaSimon Kaja, Rob C G van de Ven, J Gert van Dijk, et al.
Science Translational Medicine|March 1, 2019
An automated hybrid bioelectronic system for autogenous restoration of sinus rhythm in atrial fibrillationEmile C A Nyns, René H Poelma, Linda Volkers, et al.
Proceedings of the National Academy of Sciences of the United States of America|March 23, 2021
Functional monovalency amplifies the pathogenicity of anti-MuSK IgG4 in myasthenia gravisDana L E Vergoossen, Jaap J Plomp, Christoph Gstöttner, et al.
Pageof 7