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Proceedings of the National Academy of Sciences of the United States of America|March 21, 2002
DNA-dependent protein kinase suppresses double-strand break-induced and spontaneous homologous recombinationChris Allen, Akihiro Kurimasa, Mark A Brenneman, et al.Journal of the National Cancer Institute|May 19, 2017
Drugging the Cancers Addicted to DNA RepairJac A Nickoloff, Dennie Jones, Suk-Hee Lee, et al.Molecular Cancer Therapeutics|January 8, 2017
TAS-116, a Novel Hsp90 Inhibitor, Selectively Enhances Radiosensitivity of Human Cancer Cells to X-rays and Carbon Ion RadiationYounghyun Lee, Shigeaki Sunada, Hirokazu Hirakawa, et al.Molecular Cell|August 23, 2002
XRCC3 controls the fidelity of homologous recombination: roles for XRCC3 in late stages of recombinationMark A Brenneman, Brant M Wagener, Cheryl A Miller, et al.Frontiers in Oncology|February 14, 2022
Metnase and EEPD1: DNA Repair Functions and Potential Targets in Cancer TherapyJac A Nickoloff, Neelam Sharma, Lynn Taylor, et al.Nucleic Acids Research|June 28, 2002
Spontaneous and double-strand break-induced recombination, and gene conversion tract lengths, are differentially affected by overexpression of wild-type or ATPase-defective yeast Rad54Perry M Kim, Kimberly S Paffett, Jachen A Solinger, et al.The Journal of Biological Chemistry|December 16, 2016
Metnase Mediates Loading of Exonuclease 1 onto Single Strand Overhang DNA for End Resection at Stalled Replication ForksHyun-Suk Kim, Elizabeth A Williamson, Jac A Nickoloff, et al.DNA Repair|December 20, 2008
INO80-dependent chromatin remodeling regulates early and late stages of mitotic homologous recombinationToyoko Tsukuda, Yi-Chen Lo, Sanchita Krishna, et al.Molecular and Cellular Biology|February 17, 2005
The BRCA2-interacting protein BCCIP functions in RAD51 and BRCA2 focus formation and homologous recombinational repairHuimei Lu, Xu Guo, Xiangbing Meng, et al.DNA Repair|June 5, 2004
The kinase activity of DNA-PK is required to protect mammalian telomeresSusan M Bailey, Mark A Brenneman, James Halbrook, et al.Pageof 8