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Iscience|February 22, 2024
A safety screening platform for individualized cardiotoxicity assessmentVerena Schwach, Rolf H Slaats, Carla Cofiño-Fabres, et al.Cell Host & Microbe|June 2, 2015
Salmonella Manipulation of Host Signaling Pathways Provokes Cellular Transformation Associated with Gallbladder CarcinomaTiziana Scanu, Robbert M Spaapen, Jeroen M Bakker, et al.Pediatric Blood & Cancer|November 27, 2025
Determining Preclinical Safety of Aclarubicin in Pediatric MalignanciesDarleen S Tu, Aaron K Olson, Kimberly S Waggie, et al.Biorxiv : the Preprint Server for Biology|June 6, 2025
Determining preclinical safety of Aclarubicin in pediatric malignanciesDarleen S Tu, Aaron K Olson, Kimberly S Waggie, et al.Cell Reports|November 3, 2024
Multi-omics analyses of cancer-linked clinical salmonellae reveal bacterial-induced host metabolic shift and mTOR-dependent cell transformationVirginie Stévenin, Claudia E Coipan, Janneke W Duijster, et al.Proceedings of the National Academy of Sciences of the United States of America|September 25, 2003
Interference with T cell receptor-HLA-DR interactions by Epstein-Barr virus gp42 results in reduced T helper cell recognitionMaaike E Ressing, Daphne van Leeuwen, Frank A W Verreck, et al.Proceedings of the National Academy of Sciences of the United States of America|March 29, 2005
Varicelloviruses avoid T cell recognition by UL49.5-mediated inactivation of the transporter associated with antigen processingDanijela Koppers-Lalic, Eric A J Reits, Maaike E Ressing, et al.Journal of the American Chemical Society|September 23, 2017
A Fluorescence Polarization Activity-Based Protein Profiling Assay in the Discovery of Potent, Selective Inhibitors for Human Nonlysosomal GlucosylceramidaseDaniël Lahav, Bing Liu, Richard J B H N van den Berg, et al.Chemistry & Biology|November 23, 2006
A fluorescent broad-spectrum proteasome inhibitor for labeling proteasomes in vitro and in vivoMartijn Verdoes, Bogdan I Florea, Victoria Menendez-Benito, et al.Brain : a Journal of Neurology|April 1, 2015
Multiple sclerosis-associated CLEC16A controls HLA class II expression via late endosome biogenesisMarvin M van Luijn, Karim L Kreft, Marlieke L Jongsma, et al.Pageof 39