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Jada Lewis

Showing results (81-90 of 91) with videos related to

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American Journal of Human Genetics|May 22, 2012
Strikingly different clinicopathological phenotypes determined by progranulin-mutation dosageKatherine R Smith, John Damiano, Silvana Franceschetti, et al.
Nature Communications|June 4, 2014
ER-mitochondria associations are regulated by the VAPB-PTPIP51 interaction and are disrupted by ALS/FTD-associated TDP-43Radu Stoica, Kurt J De Vos, Sébastien Paillusson, et al.
Human Molecular Genetics|February 14, 2004
CHIP and Hsp70 regulate tau ubiquitination, degradation and aggregationLeonard Petrucelli, Dennis Dickson, Kathryn Kehoe, et al.
Acta Neuropathologica|April 28, 2012
Neuronal sensitivity to TDP-43 overexpression is dependent on timing of inductionAshley Cannon, Baoli Yang, Joshua Knight, et al.
Acta Neuropathologica|May 14, 2013
Robust cytoplasmic accumulation of phosphorylated TDP-43 in transgenic models of tauopathyAmy K Clippinger, Simon D'Alton, Wen-Lang Lin, et al.
Neurobiology of Disease|June 20, 2026
Robust tauopathy and memory deficits in a mouse model constitutively overexpressing human P301L MAPTMatthew J Hamm, Kevin McNaught, Christopher Janus, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|February 18, 2026
Progressive Supranuclear Palsy PERK haplotype B selectively translates DLX1 promoting tau toxicityChristian B Lessard, Diego Rubio Rubio, Samantha Tolton, et al.
Biorxiv : the Preprint Server for Biology|July 9, 2025
Progressive Supranuclear Palsy PERK haplotype B selectively translates DLX1 promoting tau toxicityChristian B Lessard, Diego Rubio-Rubio, Samantha Tolton, et al.
Human Molecular Genetics|February 9, 2013
Atp13a2-deficient mice exhibit neuronal ceroid lipofuscinosis, limited α-synuclein accumulation and age-dependent sensorimotor deficitsPatrick J Schultheis, Sheila M Fleming, Amy K Clippinger, et al.
Plos One|May 4, 2017
A KCNC3 mutation causes a neurodevelopmental, non-progressive SCA13 subtype associated with dominant negative effects and aberrant EGFR traffickingSwati Khare, Jerelyn A Nick, Yalan Zhang, et al.
Pageof 10

Showing results (81-90 of 91) with videos related to

Sort By:
Pageof 10
American Journal of Human Genetics|May 22, 2012
Strikingly different clinicopathological phenotypes determined by progranulin-mutation dosageKatherine R Smith, John Damiano, Silvana Franceschetti, et al.
Nature Communications|June 4, 2014
ER-mitochondria associations are regulated by the VAPB-PTPIP51 interaction and are disrupted by ALS/FTD-associated TDP-43Radu Stoica, Kurt J De Vos, Sébastien Paillusson, et al.
Human Molecular Genetics|February 14, 2004
CHIP and Hsp70 regulate tau ubiquitination, degradation and aggregationLeonard Petrucelli, Dennis Dickson, Kathryn Kehoe, et al.
Acta Neuropathologica|April 28, 2012
Neuronal sensitivity to TDP-43 overexpression is dependent on timing of inductionAshley Cannon, Baoli Yang, Joshua Knight, et al.
Acta Neuropathologica|May 14, 2013
Robust cytoplasmic accumulation of phosphorylated TDP-43 in transgenic models of tauopathyAmy K Clippinger, Simon D'Alton, Wen-Lang Lin, et al.
Neurobiology of Disease|June 20, 2026
Robust tauopathy and memory deficits in a mouse model constitutively overexpressing human P301L MAPTMatthew J Hamm, Kevin McNaught, Christopher Janus, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|February 18, 2026
Progressive Supranuclear Palsy PERK haplotype B selectively translates DLX1 promoting tau toxicityChristian B Lessard, Diego Rubio Rubio, Samantha Tolton, et al.
Biorxiv : the Preprint Server for Biology|July 9, 2025
Progressive Supranuclear Palsy PERK haplotype B selectively translates DLX1 promoting tau toxicityChristian B Lessard, Diego Rubio-Rubio, Samantha Tolton, et al.
Human Molecular Genetics|February 9, 2013
Atp13a2-deficient mice exhibit neuronal ceroid lipofuscinosis, limited α-synuclein accumulation and age-dependent sensorimotor deficitsPatrick J Schultheis, Sheila M Fleming, Amy K Clippinger, et al.
Plos One|May 4, 2017
A KCNC3 mutation causes a neurodevelopmental, non-progressive SCA13 subtype associated with dominant negative effects and aberrant EGFR traffickingSwati Khare, Jerelyn A Nick, Yalan Zhang, et al.
Pageof 10