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James D Potter

Showing results (1-10 of 53) with videos related to

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Archives of Biochemistry and Biophysics|June 6, 2022
Reprint of: The Content of Troponin, Tropomyosin, Actin, and Myosin in Rabbit Skeletal Muscle MyofibrilsJames D Potter
Results and Problems in Cell Differentiation|March 15, 2002
The role of troponin in the Ca(2+)-regulation of skeletal muscle contractionDanuta Szczesna, James D Potter
The Journal of Biological Chemistry|January 15, 2004
Familial hypertrophic cardiomyopathy mutations from different functional regions of troponin T result in different effects on the pH and Ca2+ sensitivity of cardiac muscle contractionKeita Harada, James D Potter
The Journal of Biological Chemistry|February 5, 2008
WITHDRAWN: The troponin T isoform shift may constitute a protective compensatory mechanism in hypertrophic cardiomyopathySanjeev Sirpal, James D Potter
Heart Failure Reviews|January 18, 2006
Sarcomeric protein mutations in dilated cardiomyopathyAudrey N Chang, James D Potter
Annals of the New York Academy of Sciences|June 18, 2004
Molecular and cellular aspects of troponin cardiomyopathiesAldrin V Gomes, James D Potter
Molecular and Cellular Biochemistry|November 5, 2004
Cellular and molecular aspects of familial hypertrophic cardiomyopathy caused by mutations in the cardiac troponin I geneAldrin V Gomes, James D Potter
The Journal of Biological Chemistry|September 30, 2008
Challenging current paradigms related to cardiomyopathies. Are changes in the Ca2+ sensitivity of myofilaments containing cardiac troponin C mutations (G159D and L29Q) good predictors of the phenotypic outcomes?David Dweck, Nir Hus, James D Potter
Annals of the New York Academy of Sciences|August 12, 2005
The miscommunicative cardiac cell: when good proteins go badAldrin V Gomes, Gayathri Venkatraman, James D Potter
Analytical Biochemistry|November 18, 2005
Expanding the range of free calcium regulation in biological solutionsDavid Dweck, Avelino Reyes-Alfonso, James D Potter
Pageof 6

Showing results (1-10 of 53) with videos related to

Sort By:
Pageof 6
Archives of Biochemistry and Biophysics|June 6, 2022
Reprint of: The Content of Troponin, Tropomyosin, Actin, and Myosin in Rabbit Skeletal Muscle MyofibrilsJames D Potter
Results and Problems in Cell Differentiation|March 15, 2002
The role of troponin in the Ca(2+)-regulation of skeletal muscle contractionDanuta Szczesna, James D Potter
The Journal of Biological Chemistry|January 15, 2004
Familial hypertrophic cardiomyopathy mutations from different functional regions of troponin T result in different effects on the pH and Ca2+ sensitivity of cardiac muscle contractionKeita Harada, James D Potter
The Journal of Biological Chemistry|February 5, 2008
WITHDRAWN: The troponin T isoform shift may constitute a protective compensatory mechanism in hypertrophic cardiomyopathySanjeev Sirpal, James D Potter
Heart Failure Reviews|January 18, 2006
Sarcomeric protein mutations in dilated cardiomyopathyAudrey N Chang, James D Potter
Annals of the New York Academy of Sciences|June 18, 2004
Molecular and cellular aspects of troponin cardiomyopathiesAldrin V Gomes, James D Potter
Molecular and Cellular Biochemistry|November 5, 2004
Cellular and molecular aspects of familial hypertrophic cardiomyopathy caused by mutations in the cardiac troponin I geneAldrin V Gomes, James D Potter
The Journal of Biological Chemistry|September 30, 2008
Challenging current paradigms related to cardiomyopathies. Are changes in the Ca2+ sensitivity of myofilaments containing cardiac troponin C mutations (G159D and L29Q) good predictors of the phenotypic outcomes?David Dweck, Nir Hus, James D Potter
Annals of the New York Academy of Sciences|August 12, 2005
The miscommunicative cardiac cell: when good proteins go badAldrin V Gomes, Gayathri Venkatraman, James D Potter
Analytical Biochemistry|November 18, 2005
Expanding the range of free calcium regulation in biological solutionsDavid Dweck, Avelino Reyes-Alfonso, James D Potter
Pageof 6