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The Journal of Pathology|March 13, 2016
Overexpression of MYB drives proliferation of CYLD-defective cylindroma cellsNeil Rajan, Mattias K Andersson, Naomi Sinclair, et al.
Nature|February 12, 2008
Resistance to therapy caused by intragenic deletion in BRCA2Stacey L Edwards, Rachel Brough, Christopher J Lord, et al.
Modern Pathology : an Official Journal of the United States and Canadian Academy of Pathology, Inc|January 21, 2006
Distribution and significance of nerve growth factor receptor (NGFR/p75NTR) in normal, benign and malignant breast tissueJorge S Reis-Filho, Dawn Steele, Silvana Di Palma, et al.
Molecular Cancer Therapeutics|July 26, 2013
Synthetic lethal targeting of PTEN-deficient cancer cells using selective disruption of polynucleotide kinase/phosphataseTodd R Mereniuk, Mohamed A M El Gendy, Ana M Mendes-Pereira, et al.
Cancer Cell|March 16, 2010
DNA polymerases as potential therapeutic targets for cancers deficient in the DNA mismatch repair proteins MSH2 or MLH1Sarah A Martin, Nuala McCabe, Michelle Mullarkey, et al.
Plos One|February 17, 2016
CDK1 Is a Synthetic Lethal Target for KRAS Mutant TumoursSara Costa-Cabral, Rachel Brough, Asha Konde, et al.
Gynecologic Oncology|December 22, 2007
Opportunities and challenges in ovarian cancer research, a perspective from the 11th Ovarian cancer action/HHMT Forum, Lake Como, March 2007Alan Ashworth, Frances Balkwill, Robert C Bast, et al.
Oncotarget|June 2, 2015
Oncogenic KRAS sensitizes premalignant, but not malignant cells, to Noxa-dependent apoptosis through the activation of the MEK/ERK pathwayAnnalisa Conti, Maria Teresa Majorini, Richard Elliott, et al.
Cancer Cell|February 5, 2008
Identification of CDK10 as an important determinant of resistance to endocrine therapy for breast cancerElizabeth Iorns, Nicholas C Turner, Richard Elliott, et al.
Plos One|April 21, 2017
Correction: CDK1 Is a Synthetic Lethal Target for KRAS Mutant TumoursSara Costa-Cabral, Rachel Brough, Asha Konde, et al.
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