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Arteriosclerosis, Thrombosis, and Vascular Biology|April 24, 2020
Bmal1 Deletion in Myeloid Cells Attenuates Atherosclerotic Lesion Development and Restrains Abdominal Aortic Aneurysm Formation in Hyperlipidemic MiceGuangrui Yang, Jiayang Zhang, Tingting Jiang, et al.
JCI Insight|October 6, 2017
Fibroblast activation protein augments progression and metastasis of pancreatic ductal adenocarcinomaAlbert Lo, Chung-Pin Li, Elizabeth L Buza, et al.
International Journal of Radiation Oncology, Biology, Physics|October 19, 2018
Interleukin 6 Signaling Blockade Exacerbates Acute and Late Injury From Focal Intestinal IrradiationBrett I Bell, Sravya Koduri, Carlo Salas Salinas, et al.
JCI Insight|January 11, 2019
Cardiovascular protection in females linked to estrogen-dependent inhibition of arterial stiffening and macrophage MMP12Shu-Lin Liu, Anamika Bajpai, Elizabeth A Hawthorne, et al.
American Journal of Physiology. Lung Cellular and Molecular Physiology|June 13, 2019
Loss of cells expressing fibroblast activation protein has variable effects in models of TGF-β and chronic bleomycin-induced fibrosisToru Kimura, James Monslow, Astero Klampatsa, et al.
Cancer Research|June 14, 2019
Deletion of Calcineurin Promotes a Protumorigenic Fibroblast PhenotypeAllyson Lieberman, Richard Barrett, Jaewon Kim, et al.
Circulation|July 22, 2016
Cardiovascular Consequences of Prostanoid I Receptor Deletion in Microsomal Prostaglandin E Synthase-1-Deficient Hyperlipidemic MiceSoon Yew Tang, James Monslow, Gregory R Grant, et al.
Scientific Reports|November 27, 2015
Matrix metalloproteinase-12 is an essential mediator of acute and chronic arterial stiffeningShu-Lin Liu, Yong Ho Bae, Christopher Yu, et al.
Proceedings of the National Academy of Sciences of the United States of America|April 23, 2014
Myeloid cell microsomal prostaglandin E synthase-1 fosters atherogenesis in miceLihong Chen, Guangrui Yang, James Monslow, et al.
Proceedings of the National Academy of Sciences of the United States of America|November 23, 2021
Lymph node formation and B cell homeostasis require IKK-α in distinct endothelial cell-derived compartmentsKelly A McCorkell, Nipun Jayachandran, Michelle D Cully, et al.
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