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Jan B Parys

Showing results (131-140 of 159) with videos related to

Pageof 16
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Cells|September 26, 2020
Transmembrane BAX Inhibitor-1 Motif Containing Protein 5 (TMBIM5) Sustains Mitochondrial Structure, Shape, and Function by Impacting the Mitochondrial Protein Synthesis MachineryBruno Seitaj, Felicia Maull, Li Zhang, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|February 1, 2007
Biphenyl 2,3',4,5',6-pentakisphosphate, a novel inositol polyphosphate surrogate, modulates Ca2+ responses in rat hepatocytesFabrice Vandeput, Laurent Combettes, Stephen J Mills, et al.
Cell Calcium|July 23, 2013
Bax Inhibitor-1-mediated Ca2+ leak is decreased by cytosolic acidosisSanteri Kiviluoto, Tomas Luyten, Lars Schneider, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 6, 2008
Phosphorylation of inositol 1,4,5-trisphosphate receptors by protein kinase B/Akt inhibits Ca2+ release and apoptosisTania Szado, Veerle Vanderheyden, Jan B Parys, et al.
Proceedings of the National Academy of Sciences of the United States of America|August 27, 2009
The BH4 domain of Bcl-2 inhibits ER calcium release and apoptosis by binding the regulatory and coupling domain of the IP3 receptorYi-Ping Rong, Geert Bultynck, Ademuyiwa S Aromolaran, et al.
Cell Death & Disease|July 15, 2023
LRRK2 phosphorylation status and kinase activity regulate (macro)autophagy in a Rab8a/Rab10-dependent mannerElżbieta Kania, Jaclyn S Long, David G McEwan, et al.
The FEBS Journal|November 14, 2017
A double point mutation at residues Ile14 and Val15 of Bcl-2 uncovers a role for the BH4 domain in both protein stability and functionGiovanni Monaco, Rita La Rovere, Spyridoula Karamanou, et al.
The Journal of Biological Chemistry|February 15, 2015
The BH4 domain of anti-apoptotic Bcl-XL, but not that of the related Bcl-2, limits the voltage-dependent anion channel 1 (VDAC1)-mediated transfer of pro-apoptotic Ca2+ signals to mitochondriaGiovanni Monaco, Elke Decrock, Nir Arbel, et al.
Oncotarget|August 6, 2016
The trans-membrane domain of Bcl-2α, but not its hydrophobic cleft, is a critical determinant for efficient IP3 receptor inhibitionHristina Ivanova, Abigael Ritaine, Larry Wagner, et al.
Biochemical and Biophysical Research Communications|November 8, 2012
Neuronal overexpression of IP₃ receptor 2 is detrimental in mutant SOD1 miceKim A Staats, Elke Bogaert, Nicole Hersmus, et al.
Pageof 16

Showing results (131-140 of 159) with videos related to

Sort By:
Pageof 16
Cells|September 26, 2020
Transmembrane BAX Inhibitor-1 Motif Containing Protein 5 (TMBIM5) Sustains Mitochondrial Structure, Shape, and Function by Impacting the Mitochondrial Protein Synthesis MachineryBruno Seitaj, Felicia Maull, Li Zhang, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|February 1, 2007
Biphenyl 2,3',4,5',6-pentakisphosphate, a novel inositol polyphosphate surrogate, modulates Ca2+ responses in rat hepatocytesFabrice Vandeput, Laurent Combettes, Stephen J Mills, et al.
Cell Calcium|July 23, 2013
Bax Inhibitor-1-mediated Ca2+ leak is decreased by cytosolic acidosisSanteri Kiviluoto, Tomas Luyten, Lars Schneider, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 6, 2008
Phosphorylation of inositol 1,4,5-trisphosphate receptors by protein kinase B/Akt inhibits Ca2+ release and apoptosisTania Szado, Veerle Vanderheyden, Jan B Parys, et al.
Proceedings of the National Academy of Sciences of the United States of America|August 27, 2009
The BH4 domain of Bcl-2 inhibits ER calcium release and apoptosis by binding the regulatory and coupling domain of the IP3 receptorYi-Ping Rong, Geert Bultynck, Ademuyiwa S Aromolaran, et al.
Cell Death & Disease|July 15, 2023
LRRK2 phosphorylation status and kinase activity regulate (macro)autophagy in a Rab8a/Rab10-dependent mannerElżbieta Kania, Jaclyn S Long, David G McEwan, et al.
The FEBS Journal|November 14, 2017
A double point mutation at residues Ile14 and Val15 of Bcl-2 uncovers a role for the BH4 domain in both protein stability and functionGiovanni Monaco, Rita La Rovere, Spyridoula Karamanou, et al.
The Journal of Biological Chemistry|February 15, 2015
The BH4 domain of anti-apoptotic Bcl-XL, but not that of the related Bcl-2, limits the voltage-dependent anion channel 1 (VDAC1)-mediated transfer of pro-apoptotic Ca2+ signals to mitochondriaGiovanni Monaco, Elke Decrock, Nir Arbel, et al.
Oncotarget|August 6, 2016
The trans-membrane domain of Bcl-2α, but not its hydrophobic cleft, is a critical determinant for efficient IP3 receptor inhibitionHristina Ivanova, Abigael Ritaine, Larry Wagner, et al.
Biochemical and Biophysical Research Communications|November 8, 2012
Neuronal overexpression of IP₃ receptor 2 is detrimental in mutant SOD1 miceKim A Staats, Elke Bogaert, Nicole Hersmus, et al.
Pageof 16