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Journal of Neuroimmunology|December 13, 2006
Complement activation in Lyme neuroborreliosis--increased levels of C1q and C3a in cerebrospinal fluid indicate complement activation in the CNSAnna J Henningsson, Jan Ernerudh, Kerstin Sandholm, et al.Atherosclerosis|December 11, 2007
NK cell apoptosis in coronary artery disease: relation to oxidative stressWei Li, Caroline Lidebjer, Xi-Ming Yuan, et al.The Journals of Gerontology. Series A, Biological Sciences and Medical Sciences|June 24, 2005
An immune risk phenotype, cognitive impairment, and survival in very late life: impact of allostatic load in Swedish octogenarian and nonagenarian humansAnders Wikby, Frederick Ferguson, Rosalyn Forsey, et al.American Journal of Reproductive Immunology (New York, N.Y. : 1989)|June 19, 2002
Deficiency of decidual IL-10 in first trimester missed abortion: a lack of correlation with the decidual immune cell profileMichael Plevyak, Nazeeh Hanna, Sandra Mayer, et al.Pediatric Allergy and Immunology : Official Publication of the European Society of Pediatric Allergy and Immunology|March 10, 2020
Pre- and postnatal Lactobacillus reuteri treatment alters DNA methylation of infant T helper cellsAnna Forsberg, Johanna Huoman, Simon Söderholm, et al.Mechanisms of Ageing and Development|June 6, 2006
The immune risk phenotype is associated with IL-6 in the terminal decline stage: findings from the Swedish NONA immune longitudinal study of very late life functioningAnders Wikby, Bengt-Olof Nilsson, Rosalyn Forsey, et al.International Immunology|September 24, 2005
Up-regulation of Borrelia-specific IL-4- and IFN-gamma-secreting cells in cerebrospinal fluid from children with Lyme neuroborreliosisMona Widhe, Barbro Hedin Skogman, Sara Jarefors, et al.Toxicologic Pathology|April 4, 2013
The placenta in toxicology. Part I: Animal models in toxicology: placental morphology and tolerance molecules in the cynomolgus monkey (Macaca fascicularis)Eberhard Buse, Jan-Dirk Häeger, Judit Svensson-Arvelund, et al.Nature Communications|September 5, 2023
Cis P-tau is a central circulating and placental etiologic driver and therapeutic target of preeclampsiaSukanta Jash, Sayani Banerjee, Shibin Cheng, et al.Journal of Immunology (Baltimore, Md. : 1950)|January 7, 2015
The human fetal placenta promotes tolerance against the semiallogeneic fetus by inducing regulatory T cells and homeostatic M2 macrophagesJudit Svensson-Arvelund, Ratnesh B Mehta, Robert Lindau, et al.Pageof 15