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The Canadian Journal of Cardiology|June 2, 2019
ABCA1 Agonist Mimetic Peptide CS-6253 Induces Microparticles Release From Different Cell Types by ABCA1-Efflux-Dependent MechanismAnouar Hafiane, Jan O Johansson, Jacques GenestBiochimica Et Biophysica Acta|August 6, 2014
Apolipoprotein E derived HDL mimetic peptide ATI-5261 promotes nascent HDL formation and reverse cholesterol transport in vitroAnouar Hafiane, John K Bielicki, Jan O Johansson, et al.Plos One|July 25, 2015
Novel Apo E-Derived ABCA1 Agonist Peptide (CS-6253) Promotes Reverse Cholesterol Transport and Induces Formation of preβ-1 HDL In VitroAnouar Hafiane, John K Bielicki, Jan O Johansson, et al.Biomolecules|February 27, 2026
ABCA1: A Therapeutic Target for Improving Cholesterol Homeostasis in Peripheral NeuropathiesYeon Hwa Woo, Natalie E Schmidt, Jan O Johansson, et al.Journal of Alzheimer'S Disease : JAD|August 29, 2016
ABCA1 Agonist Reverses the ApoE4-Driven Cognitive and Brain PathologiesAnat Boehm-Cagan, Roni Bar, Ori Liraz, et al.Plos One|November 9, 2016
Differential Effects of apoE4 and Activation of ABCA1 on Brain and Plasma LipoproteinsAnat Boehm-Cagan, Roni Bar, Dror Harats, et al.Molecular and Cellular Endocrinology|October 6, 2018
Novel ABCA1 peptide agonists with antidiabetic actionSalman Azhar, Stefanie Bittner, Jie Hu, et al.American Journal of Cardiovascular Drugs : Drugs, Devices, and Other Interventions|October 14, 2017
Selective BET Protein Inhibition with Apabetalone and Cardiovascular Events: A Pooled Analysis of Trials in Patients with Coronary Artery DiseaseStephen J Nicholls, Kausik K Ray, Jan O Johansson, et al.Alzheimer'S Research & Therapy|December 16, 2023
A novel apoE-mimetic increases brain apoE levels, reduces Aβ pathology and improves memory when treated before onset of pathology in male mice that express APOE3Ana C Valencia-Olvera, Deebika Balu, Shreya Bellur, et al.Proceedings of the National Academy of Sciences of the United States of America|December 24, 2021
Neuronal ROS-induced glial lipid droplet formation is altered by loss of Alzheimer's disease-associated genesMatthew J Moulton, Scott Barish, Isha Ralhan, et al.Pageof 4