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Hypertension (Dallas, Tex. : 1979)|December 3, 2019
Ang II (Angiotensin II) Conversion to Angiotensin-(1-7) in the Circulation Is POP (Prolyloligopeptidase)-Dependent and ACE2 (Angiotensin-Converting Enzyme 2)-IndependentPeter Serfozo, Jan Wysocki, Gvantca Gulua, et al.Kidney International|December 14, 2020
Knockout of aminopeptidase A in mice causes functional alterations and morphological glomerular basement membrane changes in the kidneysBenedikt Marahrens, Arndt Schulze, Jan Wysocki, et al.Kidney International|September 24, 2019
Angiotensin-(1-7) for diabetic kidney disease: better than an angiotensin-converting enzyme inhibitor alone?Alonso Marquez, Daniel BatlleAdvances in Chronic Kidney Disease|August 25, 2018
Hyperkalemic Forms of Renal Tubular Acidosis: Clinical and Pathophysiological AspectsDaniel Batlle, Jose ArrudaKidney Research and Clinical Practice|September 3, 2019
Proximal renal tubular acidosis with and without Fanconi syndromeIbrahim Kashoor, Daniel BatllePhysiological Reports|October 13, 2019
Urinary angiotensinogen antedates the development of stage 3 CKD in patients with type 1 diabetes mellitusSheeba Ba Aqeel, Minghao Ye, Jan Wysocki, et al.Hypertension (Dallas, Tex. : 1979)|May 14, 2019
Urinary Renin in Patients and Mice With Diabetic Kidney DiseaseJeannette Tang, Jan Wysocki, Minghao Ye, et al.Nephrology, Dialysis, Transplantation : Official Publication of the European Dialysis and Transplant Association - European Renal Association|November 2, 2012
Genetic causes and mechanisms of distal renal tubular acidosisDaniel Batlle, Syed K HaqueCurrent Opinion in Investigational Drugs (London, England : 2000)|February 24, 2010
Apelin and ACE2 in cardiovascular diseaseAnastasia Z Kalea, Daniel BatlleAdvances in Chronic Kidney Disease|August 25, 2018
Hypokalemic Distal Renal Tubular AcidosisPatricia G Vallés, Daniel BatllePageof 13