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BMC Microbiology|October 29, 2014
Differences in carbon source utilisation distinguish Campylobacter jejuni from Campylobacter coliSariqa Wagley, Jane Newcombe, Emma Laing, et al.
Molecular Systems Biology|January 27, 2023
One-shot 13 C15 N-metabolic flux analysis for simultaneous quantification of carbon and nitrogen fluxKhushboo Borah Slater, Martin Beyß, Ye Xu, et al.
ACS Infectious Diseases|December 28, 2020
Structural and Molecular Dynamics of Mycobacterium tuberculosis Malic Enzyme, a Potential Anti-TB Drug TargetKalistyn H Burley, Bonnie J Cuthbert, Piyali Basu, et al.
Biorxiv : the Preprint Server for Biology|November 24, 2025
VadK, a Non-Canonical Kinase that Regulates the Methylcitrate Cycle and is Essential for Mycobacterium tuberculosis VirulenceJordan Pascoe, Jane Newcombe, Jessica Mendoza, et al.
Biorxiv : the Preprint Server for Biology|March 3, 2023
Mycolactone causes destructive Sec61-dependent loss of the endothelial glycocalyx and vessel basement membrane: a new indirect mechanism driving tissue necrosis in Mycobacterium ulcerans infectionLouise Tzung-Harn Hsieh, Belinda S Hall, Jane Newcombe, et al.
Elife|February 6, 2025
The Mycobacterium ulcerans toxin mycolactone causes destructive Sec61-dependent loss of the endothelial glycocalyx and vessel basement membrane to drive skin necrosisLouise Tzung-Harn Hsieh, Belinda S Hall, Jane Newcombe, et al.
EMBO Reports|June 10, 2026
Mycobacterium tuberculosis VadK is required for the regulation of the methylcitrate cycle and virulenceJordan Pascoe, Jane Newcombe, Jessica Mendoza, et al.
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