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Experimental Neurology|June 11, 2014
Pathogenesis/genetics of frontotemporal dementia and how it relates to ALSJanis Bennion Callister, Stuart M Pickering-BrownThe European Journal of Neuroscience|June 17, 2016
Identification of biological pathways regulated by PGRN and GRN peptide treatments using transcriptome analysisSara Rollinson, Kate Young, Janis Bennion-Callister, et al.Human Molecular Genetics|November 1, 2016
Modelling C9orf72 dipeptide repeat proteins of a physiologically relevant sizeJanis Bennion Callister, Sarah Ryan, Joan Sim, et al.Human Molecular Genetics|March 13, 2018
Expression of C9orf72-related dipeptides impairs motor function in a vertebrate modelAmrutha Swaminathan, Marilou Bouffard, Meijiang Liao, et al.Neurobiology of Aging|December 1, 2014
p62/SQSTM1 analysis in frontotemporal lobar degenerationLouise Miller, Sara Rollinson, Janis Bennion Callister, et al.Acta Neuropathologica Communications|November 21, 2013
Dipeptide repeat proteins are present in the p62 positive inclusions in patients with frontotemporal lobar degeneration and motor neurone disease associated with expansions in C9ORF72David M A Mann, Sara Rollinson, Andrew Robinson, et al.Neurobiology of Aging|March 14, 2012
Analysis of the hexanucleotide repeat in C9ORF72 in Alzheimer's diseaseSara Rollinson, Nicola Halliwell, Kate Young, et al.Neurobiology of Aging|January 18, 2015
Small deletion in C9orf72 hides a proportion of expansion carriers in FTLDSara Rollinson, Janis Bennion Callister, Kate Young, et al.Neuron|September 28, 2011
A hexanucleotide repeat expansion in C9ORF72 is the cause of chromosome 9p21-linked ALS-FTDAlan E Renton, Elisa Majounie, Adrian Waite, et al.Pageof 1