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Histochemistry and Cell Biology|March 2, 2022
Localization of aquaglyceroporins in human and murine white adipose tissueFrancesco Maria Iena, Joanna Kalucka, Lærke Nielsen, et al.
Nephrology, Dialysis, Transplantation : Official Publication of the European Dialysis and Transplant Association - European Renal Association|November 22, 2007
Aldosterone-mediated apical targeting of ENaC subunits is blunted in rats with streptozotocin-induced diabetes mellitusHeidi O'Neill, Janne Lebeck, Patrick B Collins, et al.
Acta Diabetologica|September 25, 2012
Gender-specific effect of physical training on AQP7 protein expression in human adipose tissueJanne Lebeck, Torben Østergård, Aleksandra Rojek, et al.
American Journal of Physiology. Gastrointestinal and Liver Physiology|November 25, 2011
Estrogen prevents increased hepatic aquaporin-9 expression and glycerol uptake during starvationJanne Lebeck, Patrizia Gena, Heidi O'Neill, et al.
Basic & Clinical Pharmacology & Toxicology|April 2, 2014
At the centennial of Michaelis and Menten, competing Michaelis-Menten steps explain effect of GLP-1 on blood-brain transfer and metabolism of glucoseMichael Gejl, Jørgen Rungby, Birgitte Brock, et al.
Basic & Clinical Pharmacology & Toxicology|December 1, 2004
Glucagon-like peptide-1: regulation of insulin secretion and therapeutic potentialJesper Gromada, Birgitte Brock, Ole Schmitz, et al.
American Journal of Physiology. Renal Physiology|November 2, 2006
AQP7 is localized in capillaries of adipose tissue, cardiac and striated muscle: implications in glycerol metabolismMariusz T Skowronski, Janne Lebeck, Aleksandra Rojek, et al.
Ugeskrift for Laeger|May 30, 2006
[The place of Glitazones in the treatment of diabetes: after the PROactive study]Ole E Schmitz, Birgitte Brock, Jørgen Rungby, et al.
Expert Opinion on Pharmacotherapy|July 21, 2010
Differences between long-acting insulins for the treatment of type 2 diabetesMichael Gejl Jensen, Mikkel Hansen, Birgitte Brock, et al.
Metabolism: Clinical and Experimental|May 31, 2002
Glucose desensitization in INS-1 cells: evidence of impaired function caused by glucose metabolite(s) rather than by the glucose molecule per seBirgitte Brock, Jane H Mogensen, Søren Gregersen, et al.
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