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Frontiers in Physiology|November 20, 2012
The force-temperature relationship in healthy and dystrophic mouse diaphragm; implications for translational study designJason D Murray, Benjamin D Canan, Christopher D Martin, et al.
Human Molecular Genetics|August 16, 2015
Low levels of Survival Motor Neuron protein are sufficient for normal muscle function in the SMNΔ7 mouse model of SMAChitra C Iyer, Vicki L McGovern, Jason D Murray, et al.
Journal of Molecular and Cellular Cardiology|December 5, 2022
dATP elevation induces myocardial metabolic remodeling to support improved cardiac functionKetaki N Mhatre, Jason D Murray, Galina Flint, et al.
Proceedings of the National Academy of Sciences of the United States of America|May 22, 2019
Cardiac myosin activation with 2-deoxy-ATP via increased electrostatic interactions with actinJoseph D Powers, Chen-Ching Yuan, Kimberly J McCabe, et al.
Circulation|July 20, 2011
Early treatment with lisinopril and spironolactone preserves cardiac and skeletal muscle in Duchenne muscular dystrophy miceJill A Rafael-Fortney, Neeraj S Chimanji, Kevin E Schill, et al.
Journal of Molecular and Cellular Cardiology|March 26, 2021
Impact of etiology on force and kinetics of left ventricular end-stage failing human myocardiumMohammed A Mashali, Nancy S Saad, Benjamin D Canan, et al.
Journal of Molecular and Cellular Cardiology|July 9, 2018
Etiology-dependent impairment of relaxation kinetics in right ventricular end-stage failing human myocardiumJae-Hoon Chung, Brit L Martin, Benjamin D Canan, et al.
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