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Communicative & Integrative Biology|July 31, 2009
Ion channels as effectors in carbon monoxide signalingChris Peers, Mark L Dallas, Jason L ScraggNeuroreport|February 28, 2008
Modulation of hTREK-1 by carbon monoxideMark L Dallas, Jason L Scragg, Chris PeersCell Calcium|December 19, 2006
Molecular requirements for L-type Ca2+ channel blockade by testosteroneJason L Scragg, Mark L Dallas, Chris PeersBiochemical and Biophysical Research Communications|May 11, 2005
Constitutive activity of endogenous receptors by inducible Gq overexpressionJason L Scragg, Stephen G Ball, Anthony J BalmforthBiochemical and Biophysical Research Communications|July 5, 2005
Constitutive activity of human angiotensin II type-1 receptors by Gq overexpressionJason L Scragg, Philip Warburton, Stephen G Ball, et al.Biochemical and Biophysical Research Communications|April 29, 2015
H2S does not regulate proliferation via T-type Ca2+ channelsJacobo Elies, Emily Johnson, John P Boyle, et al.The Journal of Physiology|January 26, 2016
Regulation of the T-type Ca(2+) channel Cav3.2 by hydrogen sulfide: emerging controversies concerning the role of H2 S in nociceptionJacobo Elies, Jason L Scragg, John P Boyle, et al.Pflugers Archiv : European Journal of Physiology|November 29, 2007
H2O 2-stimulated Ca2+ influx via TRPM2 is not the sole determinant of subsequent cell deathJenny A Wilkinson, Jason L Scragg, John P Boyle, et al.The Journal of Biological Chemistry|July 4, 2008
Carbon monoxide inhibits L-type Ca2+ channels via redox modulation of key cysteine residues by mitochondrial reactive oxygen speciesJason L Scragg, Mark L Dallas, Jenny A Wilkinson, et al.Antioxidants & Redox Signaling|November 15, 2011
Modulation of ion channels by hydrogen sulfideChris Peers, Claudia C Bauer, John P Boyle, et al.Pageof 4