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Jean-Jacques Martin

Showing results (61-70 of 107) with videos related to

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Alzheimer'S Research & Therapy|March 22, 2018
Diagnostic value of cerebrospinal fluid tau, neurofilament, and progranulin in definite frontotemporal lobar degenerationJoery Goossens, Maria Bjerke, Sara Van Mossevelde, et al.
Neurology. Genetics|September 27, 2016
Mutated CTSF in adult-onset neuronal ceroid lipofuscinosis and FTDJulie van der Zee, Peter Mariën, Roeland Crols, et al.
The American Journal of Pathology|August 7, 2002
Dense-core senile plaques in the Flemish variant of Alzheimer's disease are vasocentricSamir Kumar-Singh, Patrick Cras, Rong Wang, et al.
Neurobiology of Aging|September 22, 2021
Neurogranin as biomarker in CSF is non-specific to Alzheimer's disease dementiaEline A J Willemse, Anne Sieben, Charisse Somers, et al.
Journal of Alzheimer'S Disease : JAD|November 7, 2022
Improved Alzheimer's Disease versus Frontotemporal Lobar Degeneration Differential Diagnosis Combining EEG and Neurochemical Biomarkers: A Pilot StudyJorne Laton, Jeroen Van Schependom, Joery Goossens, et al.
Alzheimer'S & Dementia (Amsterdam, Netherlands)|May 22, 2018
Monoaminergic impairment in Down syndrome with Alzheimer's disease compared to early-onset Alzheimer's diseaseAlain D Dekker, Yannick Vermeiren, Maria Carmona-Iragui, et al.
Neurology|May 13, 2014
Partial deletion of AFG3L2 causing spinocerebellar ataxia type 28Katrien Smets, Tine Deconinck, Jonathan Baets, et al.
Human Mutation|February 7, 2008
Null mutations causing depletion of the type 1 ryanodine receptor (RYR1) are commonly associated with recessive structural congenital myopathies with coresNicole Monnier, Isabelle Marty, Julien Faure, et al.
Journal of Alzheimer'S Disease : JAD|September 17, 2016
EEG Dominant Frequency Peak Differentiates Between Alzheimer's Disease and Frontotemporal Lobar DegenerationJoery Goossens, Jorne Laton, Jeroen Van Schependom, et al.
American Journal of Human Genetics|August 23, 2002
Mutations of the selenoprotein N gene, which is implicated in rigid spine muscular dystrophy, cause the classical phenotype of multiminicore disease: reassessing the nosology of early-onset myopathiesAna Ferreiro, Susana Quijano-Roy, Claire Pichereau, et al.
Pageof 11

Showing results (61-70 of 107) with videos related to

Sort By:
Pageof 11
Alzheimer'S Research & Therapy|March 22, 2018
Diagnostic value of cerebrospinal fluid tau, neurofilament, and progranulin in definite frontotemporal lobar degenerationJoery Goossens, Maria Bjerke, Sara Van Mossevelde, et al.
Neurology. Genetics|September 27, 2016
Mutated CTSF in adult-onset neuronal ceroid lipofuscinosis and FTDJulie van der Zee, Peter Mariën, Roeland Crols, et al.
The American Journal of Pathology|August 7, 2002
Dense-core senile plaques in the Flemish variant of Alzheimer's disease are vasocentricSamir Kumar-Singh, Patrick Cras, Rong Wang, et al.
Neurobiology of Aging|September 22, 2021
Neurogranin as biomarker in CSF is non-specific to Alzheimer's disease dementiaEline A J Willemse, Anne Sieben, Charisse Somers, et al.
Journal of Alzheimer'S Disease : JAD|November 7, 2022
Improved Alzheimer's Disease versus Frontotemporal Lobar Degeneration Differential Diagnosis Combining EEG and Neurochemical Biomarkers: A Pilot StudyJorne Laton, Jeroen Van Schependom, Joery Goossens, et al.
Alzheimer'S & Dementia (Amsterdam, Netherlands)|May 22, 2018
Monoaminergic impairment in Down syndrome with Alzheimer's disease compared to early-onset Alzheimer's diseaseAlain D Dekker, Yannick Vermeiren, Maria Carmona-Iragui, et al.
Neurology|May 13, 2014
Partial deletion of AFG3L2 causing spinocerebellar ataxia type 28Katrien Smets, Tine Deconinck, Jonathan Baets, et al.
Human Mutation|February 7, 2008
Null mutations causing depletion of the type 1 ryanodine receptor (RYR1) are commonly associated with recessive structural congenital myopathies with coresNicole Monnier, Isabelle Marty, Julien Faure, et al.
Journal of Alzheimer'S Disease : JAD|September 17, 2016
EEG Dominant Frequency Peak Differentiates Between Alzheimer's Disease and Frontotemporal Lobar DegenerationJoery Goossens, Jorne Laton, Jeroen Van Schependom, et al.
American Journal of Human Genetics|August 23, 2002
Mutations of the selenoprotein N gene, which is implicated in rigid spine muscular dystrophy, cause the classical phenotype of multiminicore disease: reassessing the nosology of early-onset myopathiesAna Ferreiro, Susana Quijano-Roy, Claire Pichereau, et al.
Pageof 11