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Neuroreport|September 17, 2004
A novel somatodendritic marker defined by a peptide derived from the ALS2 proteinVirginie Bros, Stephen J A Shemilt, Jonathan D Cooper, et al.Human Molecular Genetics|June 6, 2009
Correction of tau mis-splicing caused by FTDP-17 MAPT mutations by spliceosome-mediated RNA trans-splicingTeresa Rodriguez-Martin, Karen Anthony, Mariano A Garcia-Blanco, et al.Acta Neuropathologica Communications|June 26, 2015
Dipeptide repeat protein inclusions are rare in the spinal cord and almost absent from motor neurons in C9ORF72 mutant amyotrophic lateral sclerosis and are unlikely to cause their degenerationJorge Gomez-Deza, Youn-Bok Lee, Claire Troakes, et al.Neurobiology of Aging|October 29, 2015
Lack of association between TDP-43 pathology and tau mis-splicing in Alzheimer's diseaseMichael Niblock, Tibor Hortobágyi, Claire Troakes, et al.EMBO Reports|September 15, 2020
Loss of TMEM106B potentiates lysosomal and FTLD-like pathology in progranulin-deficient miceGeorg Werner, Markus Damme, Martin Schludi, et al.Cell Motility and the Cytoskeleton|February 18, 2003
Dynamic properties of APC-decorated microtubules in living cellsRejith Dayanandan, Rachel Butler, Phillip R Gordon-Weeks, et al.The Journal of Biological Chemistry|August 27, 2021
The porphyrin TMPyP4 inhibits elongation during the noncanonical translation of the FTLD/ALS-associated GGGGCC repeat in the C9orf72 geneKohji Mori, Shiho Gotoh, Tomoko Yamashita, et al.Acta Neuropathologica|October 8, 2013
Dipeptide repeat protein pathology in C9ORF72 mutation cases: clinico-pathological correlationsIan R Mackenzie, Thomas Arzberger, Elisabeth Kremmer, et al.Nature Neuroscience|September 6, 2011
miR-132, an experience-dependent microRNA, is essential for visual cortex plasticityNikolaos Mellios, Hiroki Sugihara, Jorge Castro, et al.EMBO Reports|July 28, 2016
Reduced hnRNPA3 increases C9orf72 repeat RNA levels and dipeptide-repeat protein depositionKohji Mori, Yoshihiro Nihei, Thomas Arzberger, et al.Pageof 14