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Human Molecular Genetics|March 6, 2013
Trans-splicing correction of tau isoform imbalance in a mouse model of tau mis-splicingMaría Elena Avale, Teresa Rodríguez-Martín, Jean-Marc GalloActa Neuropathologica|December 25, 2013
Modelling C9ORF72 hexanucleotide repeat expansion in amyotrophic lateral sclerosis and frontotemporal dementiaAlan Stepto, Jean-Marc Gallo, Christopher E Shaw, et al.Neuroscience Letters|October 9, 2008
Focal distortion of the nuclear envelope by huntingtin aggregates revealed by lamin immunostainingJ Paul Chapple, Virginie Bros-Facer, Rachel Butler, et al.Frontiers in Cellular Neuroscience|July 27, 2017
RNA Misprocessing in C9orf72-Linked NeurodegenerationHolly V Barker, Michael Niblock, Youn-Bok Lee, et al.Neuroreport|November 20, 2002
Ligand-dependent aggregation of polyglutamine-expanded androgen receptor in neuronal cellsR Siobhan Darrington, Rachel Butler, P Nigel Leigh, et al.Human Molecular Genetics|August 30, 2007
Expression, localization and tau exon 10 splicing activity of the brain RNA-binding protein TNRC4J Paul Chapple, Karen Anthony, Teresa Rodriguez Martin, et al.Neuroreport|September 17, 2004
A novel somatodendritic marker defined by a peptide derived from the ALS2 proteinVirginie Bros, Stephen J A Shemilt, Jonathan D Cooper, et al.Human Molecular Genetics|June 6, 2009
Correction of tau mis-splicing caused by FTDP-17 MAPT mutations by spliceosome-mediated RNA trans-splicingTeresa Rodriguez-Martin, Karen Anthony, Mariano A Garcia-Blanco, et al.Acta Neuropathologica Communications|June 26, 2015
Dipeptide repeat protein inclusions are rare in the spinal cord and almost absent from motor neurons in C9ORF72 mutant amyotrophic lateral sclerosis and are unlikely to cause their degenerationJorge Gomez-Deza, Youn-Bok Lee, Claire Troakes, et al.Neurobiology of Aging|October 29, 2015
Lack of association between TDP-43 pathology and tau mis-splicing in Alzheimer's diseaseMichael Niblock, Tibor Hortobágyi, Claire Troakes, et al.Pageof 4