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Cell Adhesion & Migration|April 20, 2010
What is the role of amyloid precursor protein dimerization?Naouel Ben Khalifa, Joanne Van Hees, Bernadette Tasiaux, et al.
Neurobiology of Aging|May 4, 2017
Amyloid precursor protein reduction enhances the formation of neurofibrillary tangles in a mutant tau transgenic mouse modelVirginie Vanden Dries, Virginie Stygelbout, Nathalie Pierrot, et al.
The American Journal of Pathology|October 3, 2012
Lack of tau proteins rescues neuronal cell death and decreases amyloidogenic processing of APP in APP/PS1 miceKarelle Leroy, Kunie Ando, Vincent Laporte, et al.
Frontiers in Molecular Neuroscience|May 11, 2023
Conditional deletion of KCC2 impairs synaptic plasticity and both spatial and nonspatial memoryAnna Kreis, Farah Issa, Xavier Yerna, et al.
Neurobiology of Disease|October 1, 2013
Increased misfolding and truncation of tau in APP/PS1/tau transgenic mice compared to mutant tau miceCéline Héraud, Doris Goufak, Kunie Ando, et al.
The Journal of Biological Chemistry|January 24, 2015
Presenilin transmembrane domain 8 conserved AXXXAXXXG motifs are required for the activity of the γ-secretase complexClaudia Marinangeli, Bernadette Tasiaux, Rémi Opsomer, et al.
Acta Neuropathologica|February 6, 2019
Aggregated Tau activates NLRP3-ASC inflammasome exacerbating exogenously seeded and non-exogenously seeded Tau pathology in vivoIlie-Cosmin Stancu, Niels Cremers, Hannah Vanrusselt, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|November 17, 2011
Structural features of the KPI domain control APP dimerization, trafficking, and processingNaouel Ben Khalifa, Donatienne Tyteca, Claudia Marinangeli, et al.
Frontiers in Physiology|November 1, 2017
Presenilin 2-Dependent Maintenance of Mitochondrial Oxidative Capacity and MorphologySabrina Contino, Paolo E Porporato, Matthew Bird, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|January 1, 2016
APP-dependent glial cell line-derived neurotrophic factor gene expression drives neuromuscular junction formationSerena Stanga, Nadège Zanou, Emilie Audouard, et al.
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