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Nature Reviews. Drug Discovery|April 12, 2008
The TWEAK-Fn14 cytokine-receptor axis: discovery, biology and therapeutic targetingJeffrey A WinklesDrug News & Perspectives|February 15, 2007
Inhibition of TWEAK activity as a new treatment for inflammatory and degenerative diseasesManuel Yepes, Jeffrey A WinklesOncogene|January 11, 2005
Differential regulation of polo-like kinase 1, 2, 3, and 4 gene expression in mammalian cells and tissuesJeffrey A Winkles, Gregory F AlbertsCell Cycle (Georgetown, Tex.)|April 27, 2004
Murine FGF-inducible kinase is rapidly degraded via the nuclear ubiquitin-proteosome system when overexpressed in NIH 3T3 cellsGregory F Alberts, Jeffrey A WinklesCytokine & Growth Factor Reviews|June 6, 2003
TWEAK, a member of the TNF superfamily, is a multifunctional cytokine that binds the TweakR/Fn14 receptorSteven R Wiley, Jeffrey A WinklesCells, Tissues, Organs|August 30, 2008
Progressive ankylosis gene (ank) regulates osteoblast differentiationThorsten Kirsch, Hyon Jong Kim, Jeffrey A WinklesCancer Letters|May 12, 2005
TWEAK and Fn14: new molecular targets for cancer therapy?Jeffrey A Winkles, Nhan L Tran, Michael E BerensThe Journal of Biological Chemistry|April 14, 2010
Full-length, membrane-anchored TWEAK can function as a juxtacrine signaling molecule and activate the NF-kappaB pathwaySharron A N Brown, Arundhati Ghosh, Jeffrey A WinklesThe Journal of Biological Chemistry|March 22, 2014
Regulation of fibroblast growth factor-inducible 14 (Fn14) expression levels via ligand-independent lysosomal degradationSujatha Gurunathan, Jeffrey A Winkles, Sankar Ghosh, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|November 4, 2005
Tumor necrosis factor-like weak inducer of apoptosis increases the permeability of the neurovascular unit through nuclear factor-kappa B pathway activationRohini Polavarapu, Maria Carolina Gongora, Jeffrey A Winkles, et al.Pageof 9