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Proceedings of the National Academy of Sciences of the United States of America|March 1, 2017
Genetic variation in chromosome Y regulates susceptibility to influenza A virus infectionDimitry N Krementsov, Laure K Case, Oliver Dienz, et al.Plos One|September 27, 2013
The endogenous Th17 response in NO2-promoted allergic airway disease is dispensable for airway hyperresponsiveness and distinct from Th17 adoptive transferRebecca A Martin, Jennifer L Ather, Rebecca Daggett, et al.The Journal of Allergy and Clinical Immunology. Global|December 5, 2022
Ketone body augmentation decreases methacholine hyperresponsiveness in mouse models of allergic asthmaMadeleine M Mank, Leah F Reed, V Amanda Fastiggi, et al.Pulmonary Pharmacology & Therapeutics|May 10, 2017
The role of iNKT cells on the phenotypes of allergic airways in a mouse modelLennart K A Lundblad, Nazey Gülec, Matthew E Poynter, et al.Journal of Immunology (Baltimore, Md. : 1950)|May 31, 2011
Serum amyloid A activates the NLRP3 inflammasome and promotes Th17 allergic asthma in miceJennifer L Ather, Karina Ckless, Rebecca Martin, et al.Scientific Reports|September 22, 2019
The Effect of Flavored E-cigarettes on Murine Allergic Airways DiseaseDavid G Chapman, Dylan T Casey, Jennifer L Ather, et al.Genes and Immunity|June 9, 2018
Regulation of invariant NKT cell development and function by a 0.14 Mbp locus on chromosome 1: a possible role for Fcgr3Victoria L DeVault, Murisa Malagic, Linda Mei, et al.American Journal of Respiratory Cell and Molecular Biology|February 2, 2013
Interleukin-1 receptor and caspase-1 are required for the Th17 response in nitrogen dioxide-promoted allergic airway diseaseRebecca A Martin, Jennifer L Ather, Lennart K A Lundblad, et al.American Journal of Physiology. Lung Cellular and Molecular Physiology|August 14, 2020
Therapeutic efficacy of IL-17A neutralization with corticosteroid treatment in a model of antigen-driven mixed-granulocytic asthmaKatherine E Menson, Madeleine M Mank, Leah F Reed, et al.Mucosal Immunology|June 2, 2022
Macrophage-intrinsic DUOX1 contributes to type 2 inflammation and mucus metaplasia during allergic airway diseaseCarolyn R Morris, Aida Habibovic, Christopher M Dustin, et al.Pageof 5