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Jennifer L Estall

Showing results (11-20 of 56) with videos related to

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Endocrinology|June 20, 2015
To Be(ta Cell) or Not to Be(ta cell): New Mouse Models for Studying Gene Function in the Pancreatic β-CellJennifer L Estall, Robert A Screaton
Endocrinology|December 17, 2004
Tales beyond the crypt: glucagon-like peptide-2 and cytoprotection in the intestinal mucosaJennifer L Estall, Daniel J Drucker
Endocrinology|November 30, 2017
Linking Metabolic Disease With the PGC-1α Gly482Ser PolymorphismRoxanne Vandenbeek, Naveen P Khan, Jennifer L Estall
Molecular Biology of the Cell|June 1, 2004
Lipid raft-dependent glucagon-like peptide-2 receptor trafficking occurs independently of agonist-induced desensitizationJennifer L Estall, Bernardo Yusta, Daniel J Drucker
The Journal of Biological Chemistry|April 9, 2005
The glucagon-like peptide-2 receptor C terminus modulates beta-arrestin-2 association but is dispensable for ligand-induced desensitization, endocytosis, and G-protein-dependent effector activationJennifer L Estall, Jacqueline A Koehler, Bernardo Yusta, et al.
Micropublication Biology|September 9, 2024
Commercially available PGC-1α antibodies vary greatly in specificity and sensitivityMaria Galipeau, Clémence Schmitt, Cindy Baldwin, et al.
The Journal of Biological Chemistry|June 27, 2018
Peptide-based sequestration of the adaptor protein Nck1 in pancreatic β cells enhances insulin biogenesis and protects against diabetogenic stressesGeorge Kefalas, Nathalie Jouvet, Cindy Baldwin, et al.
Endocrinology|January 13, 2026
Modelling the Spectrum of Type 2 Diabetes in MiceLili Grieco-St-Pierre, Myriam P Hoyeck, Jennifer L Estall, et al.
Gastroenterology|May 12, 2005
Mucosal adaptation to enteral nutrients is dependent on the physiologic actions of glucagon-like peptide-2 in miceEric D Shin, Jennifer L Estall, Angelo Izzo, et al.
American Journal of Physiology. Endocrinology and Metabolism|September 14, 2020
Differences in metabolic and liver pathobiology induced by two dietary mouse models of nonalcoholic fatty liver diseaseHannah Zhang, Mélissa Léveillé, Emilie Courty, et al.
Pageof 6

Showing results (11-20 of 56) with videos related to

Sort By:
Pageof 6
Endocrinology|June 20, 2015
To Be(ta Cell) or Not to Be(ta cell): New Mouse Models for Studying Gene Function in the Pancreatic β-CellJennifer L Estall, Robert A Screaton
Endocrinology|December 17, 2004
Tales beyond the crypt: glucagon-like peptide-2 and cytoprotection in the intestinal mucosaJennifer L Estall, Daniel J Drucker
Endocrinology|November 30, 2017
Linking Metabolic Disease With the PGC-1α Gly482Ser PolymorphismRoxanne Vandenbeek, Naveen P Khan, Jennifer L Estall
Molecular Biology of the Cell|June 1, 2004
Lipid raft-dependent glucagon-like peptide-2 receptor trafficking occurs independently of agonist-induced desensitizationJennifer L Estall, Bernardo Yusta, Daniel J Drucker
The Journal of Biological Chemistry|April 9, 2005
The glucagon-like peptide-2 receptor C terminus modulates beta-arrestin-2 association but is dispensable for ligand-induced desensitization, endocytosis, and G-protein-dependent effector activationJennifer L Estall, Jacqueline A Koehler, Bernardo Yusta, et al.
Micropublication Biology|September 9, 2024
Commercially available PGC-1α antibodies vary greatly in specificity and sensitivityMaria Galipeau, Clémence Schmitt, Cindy Baldwin, et al.
The Journal of Biological Chemistry|June 27, 2018
Peptide-based sequestration of the adaptor protein Nck1 in pancreatic β cells enhances insulin biogenesis and protects against diabetogenic stressesGeorge Kefalas, Nathalie Jouvet, Cindy Baldwin, et al.
Endocrinology|January 13, 2026
Modelling the Spectrum of Type 2 Diabetes in MiceLili Grieco-St-Pierre, Myriam P Hoyeck, Jennifer L Estall, et al.
Gastroenterology|May 12, 2005
Mucosal adaptation to enteral nutrients is dependent on the physiologic actions of glucagon-like peptide-2 in miceEric D Shin, Jennifer L Estall, Angelo Izzo, et al.
American Journal of Physiology. Endocrinology and Metabolism|September 14, 2020
Differences in metabolic and liver pathobiology induced by two dietary mouse models of nonalcoholic fatty liver diseaseHannah Zhang, Mélissa Léveillé, Emilie Courty, et al.
Pageof 6