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Journal of Huntington'S Disease|September 24, 2016
Similar Progression of Morphological and Metabolic Phenotype in R6/2 Mice with Different CAG Repeats Revealed by In Vivo Magnetic Resonance Imaging and SpectroscopyStephen J Sawiak, Nigel I Wood, A Jennifer MortonNature Reviews. Neuroscience|September 21, 2013
Choosing an animal model for the study of Huntington's diseaseMahmoud A Pouladi, A Jennifer Morton, Michael R HaydenGlia|August 5, 2003
Microglia density decreases with age in a mouse model of Huntington's diseaseLi Ma, A Jennifer Morton, Louise F B NicholsonEneuro|November 21, 2019
Abnormal Photic Entrainment to Phase-Delaying Stimuli in the R6/2 Mouse Model of Huntington's Disease, despite Retinal Responsiveness to LightKoliane Ouk, Juliet Aungier, Michelle Ware, et al.Neurotherapeutics : the Journal of the American Society for Experimental Neurotherapeutics|June 28, 2017
Chronic Paroxetine Treatment Prevents the Emergence of Abnormal Electroencephalogram Oscillations in Huntington's Disease MiceSandor Kantor, Janos Varga, Shreya Kulkarni, et al.Brain and Behavior|October 21, 2014
Beneficial effects of environmental enrichment and food entrainment in the R6/2 mouse model of Huntington's diseaseElizabeth A Skillings, Nigel I Wood, A Jennifer MortonNeuropharmacology|December 25, 2017
Chronic paroxetine treatment prevents disruption of methamphetamine-sensitive circadian oscillator in a transgenic mouse model of Huntington's diseaseKoliane Ouk, Juliet Aungier, Marc Cuesta, et al.Journal of Neurochemistry|June 18, 2002
The role of dopamine in motor symptoms in the R6/2 transgenic mouse model of Huntington's diseaseMiriam A Hickey, Gavin P Reynolds, A Jennifer MortonBrain Research Bulletin|March 14, 2007
Depletion of Complexin II does not affect disease progression in a mouse model of Huntington's disease (HD); support for role for complexin II in behavioural pathology in a mouse model of HDDervila Glynn, Kerstin Reim, Nils Brose, et al.Brain Research Bulletin|November 18, 2008
The detection and measurement of locomotor deficits in a transgenic mouse model of Huntington's disease are task- and protocol-dependent: influence of non-motor factors on locomotor functionPatrick N Pallier, Cheney J G Drew, A Jennifer MortonPageof 16