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Biochemical Pharmacology|July 15, 2020
Recombinant production, bioconjugation and membrane binding studies ofPn3a, a selective NaV1.7 inhibitorGagan Sharma, Jennifer R Deuis, Xinying Jia, et al.Plos One|March 12, 2019
Development of a high-throughput fluorescent no-wash sodium influx assayBryan Tay, Teneale A Stewart, Felicity M Davis, et al.Molecular Immunology|December 25, 2017
Role of complement anaphylatoxin receptors in a mouse model of acute burn-induced painMichael Morgan, Jennifer R Deuis, Trent M Woodruff, et al.The Journal of Biological Chemistry|February 15, 2022
Structural and functional insights into the inhibition of human voltage-gated sodium channels by μ-conotoxin KIIIA disulfide isomersHue N T Tran, Kirsten L McMahon, Jennifer R Deuis, et al.Journal of Medicinal Chemistry|January 31, 2018
The E15R Point Mutation in Scorpion Toxin Cn2 Uncouples Its Depressant and Excitatory Activities on Human NaV1.6Mathilde R Israel, Panumart Thongyoo, Jennifer R Deuis, et al.Advances in Experimental Medicine and Biology|October 25, 2019
High-Throughput Fluorescence Assays for Ion Channels and GPCRsIrina Vetter, David Carter, John Bassett, et al.The Journal of Biological Chemistry|September 11, 2024
A venom peptide-induced NaV channel modulation mechanism involving the interplay between fixed channel charges and ionic gradientsAshvriya Thapa, Jia Hao Beh, Samuel D Robinson, et al.Biomedicines|October 7, 2020
Discovery, Pharmacological Characterisation and NMR Structure of the Novel µ-Conotoxin SxIIIC, a Potent and Irreversible NaV Channel InhibitorKirsten L McMahon, Hue N T Tran, Jennifer R Deuis, et al.Toxins|September 22, 2022
µ-Conotoxins Targeting the Human Voltage-Gated Sodium Channel Subtype NaV1.7Kirsten L McMahon, Hue N T Tran, Jennifer R Deuis, et al.Human Molecular Genetics|September 18, 2023
Erythromelalgia caused by the missense mutation p.Arg220Pro in an alternatively spliced exon of SCN9A (NaV1.7)Jennifer R Deuis, Smitha Kumble, Angelo Keramidas, et al.Pageof 7