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Cells|January 20, 2021
NMDA and AMPA Receptor Autoantibodies in Brain Disorders: From Molecular Mechanisms to Clinical FeaturesFabrizio Gardoni, Jennifer Stanic, Diego Scheggia, et al.Cells|May 28, 2022
Rabphilin-3A Drives Structural Modifications of Dendritic Spines Induced by Long-Term PotentiationLuca Franchini, Jennifer Stanic, Marta Barzasi, et al.The Journal of Biological Chemistry|April 12, 2012
N-methyl-D-aspartate (NMDA) receptor composition modulates dendritic spine morphology in striatal medium spiny neuronsCsaba Vastagh, Fabrizio Gardoni, Vincenza Bagetta, et al.Molecular Brain|May 13, 2016
LRRK2 phosphorylation level correlates with abnormal motor behaviour in an experimental model of levodopa-induced dyskinesiasJennifer Stanic, Manuela Mellone, Maria Daniela Cirnaru, et al.Brain, Behavior, and Immunity|July 11, 2021
GluA3 autoantibodies induce alterations in dendritic spine and behavior in miceDiego Scheggia, Jennifer Stanic, Maria Italia, et al.Nature Communications|December 19, 2015
Rabphilin 3A retains NMDA receptors at synaptic sites through interaction with GluN2A/PSD-95 complexJennifer Stanic, Mario Carta, Ivano Eberini, et al.The Journal of Comparative Neurology|February 27, 2016
Developmental and adult expression patterns of the G-protein-coupled receptor GPR88 in the rat: Establishment of a dual nuclear-cytoplasmic localizationRenaud Massart, Virginie Mignon, Jennifer Stanic, et al.Elife|March 6, 2018
Early structural and functional plasticity alterations in a susceptibility period of DYT1 dystonia mouse striatumMarta Maltese, Jennifer Stanic, Annalisa Tassone, et al.Frontiers in Cellular Neuroscience|July 29, 2015
NMDA receptor GluN2A/GluN2B subunit ratio as synaptic trait of levodopa-induced dyskinesias: from experimental models to patientsManuela Mellone, Jennifer Stanic, Ledia F Hernandez, et al.Neurobiology of Disease|September 28, 2018
NMDA receptor GluN2D subunit participates to levodopa-induced dyskinesia pathophysiologyManuela Mellone, Elisa Zianni, Jennifer Stanic, et al.Pageof 2