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Cells|August 12, 2022
Systematic Functional Analysis of PINK1 and PRKN Coding VariantsBenjamin J Broadway, Paige K Boneski, Jenny M Bredenberg, et al.
Brain Pathology (Zurich, Switzerland)|June 1, 2023
Alpha-synuclein-associated changes in PINK1-PRKN-mediated mitophagy are disease context dependentXu Hou, Taylor Hsuan-Yu Chen, Shunsuke Koga, et al.
Cells|September 27, 2024
Structural and Functional Characterization of the Most Frequent Pathogenic PRKN Substitution p.R275WBernardo A Bustillos, Liam T Cocker, Mathew A Coban, et al.
Autophagy|December 2, 2023
Basal activity of PINK1 and PRKN in cell models and rodent brainJens O Watzlawik, Fabienne C Fiesel, Gabriella Fiorino, et al.
Autophagy|July 8, 2025
Activation of endogenous PRKN by structural derepression is linked to increased turnover of the E3 ubiquitin ligaseFabienne C Fiesel, Bernardo A Bustillos, Jens O Watzlawik, et al.
Autophagy|December 5, 2022
Substitution of PINK1 Gly411 modulates substrate receptivity and turnoverFabienne C Fiesel, Dominika Fričová, Caleb S Hayes, et al.
Molecular Neurodegeneration|October 18, 2018
ABI3 and PLCG2 missense variants as risk factors for neurodegenerative diseases in Caucasians and African AmericansOlivia J Conway, Minerva M Carrasquillo, Xue Wang, et al.
International Journal of Molecular Sciences|July 9, 2022
Cathepsin B p.Gly284Val Variant in Parkinson's Disease PathogenesisLukasz M Milanowski, Xu Hou, Jenny M Bredenberg, et al.
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