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Jeremy A Simpson

Showing results (41-50 of 66) with videos related to

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American Journal of Hypertension|October 4, 2021
An Evaluation of Cardiac Health in the Spontaneously Hypertensive Rat Colony: Implications of Evolutionary Driven Increases in Concentric HypertrophyEmma J B Holjak, Iryna Savinova, Victoria L Nelson, et al.
Redox Biology|August 23, 2024
Mitohormesis during advanced stages of Duchenne muscular dystrophy reveals a redox-sensitive creatine pathway that can be enhanced by the mitochondrial-targeting peptide SBT-20Meghan C Hughes, Sofhia V Ramos, Aditya N Brahmbhatt, et al.
Science Translational Medicine|May 19, 2017
Central-acting therapeutics alleviate respiratory weakness caused by heart failure-induced ventilatory overdriveAndrew J Foster, Mathew J Platt, Jason S Huber, et al.
Frontiers in Physiology|July 16, 2024
Cardiomyocyte crosstalk with endothelium modulates cardiac structure, function, and ischemia-reperfusion injury susceptibility through erythropoietinJade P Marrow, Razan Alshamali, Brittany A Edgett, et al.
Chronobiology International|July 25, 2012
Chronomics of pressure overload-induced cardiac hypertrophy in mice reveals altered day/night gene expression and biomarkers of heart diseaseElena V Tsimakouridze, Marty Straume, Peter S Podobed, et al.
CJC Open|June 7, 2024
Better Respiratory Function in Heart Failure Patients With Use of Central-Acting TherapeuticsJulie K K Vishram-Nielsen, Fernando Luis Scolari, Chun-Po Steve Fan, et al.
Journal of the American Society of Nephrology : JASN|January 24, 2016
Nephrin Tyrosine Phosphorylation Is Required to Stabilize and Restore Podocyte Foot Process ArchitectureLaura A New, Claire E Martin, Rizaldy P Scott, et al.
Journal of the American College of Cardiology|May 14, 2011
The primary benefits of angiotensin-converting enzyme inhibition on cardiac remodeling occur during sleep time in murine pressure overload hypertrophyTami A Martino, Nazneen Tata, Jeremy A Simpson, et al.
Circulation. Arrhythmia and Electrophysiology|July 13, 2011
Iron overload decreases CaV1.3-dependent L-type Ca2+ currents leading to bradycardia, altered electrical conduction, and atrial fibrillationRobert A Rose, Michael Sellan, Jeremy A Simpson, et al.
Journal of the American Society of Nephrology : JASN|July 29, 2022
Complementary Nck1/2 Signaling in Podocytes Controls <b></b> Actinin-4-Mediated Actin Organization, Adhesion, and Basement Membrane CompositionClaire E Martin, Noah J Phippen, Ava Keyvani Chahi, et al.
Pageof 7

Showing results (41-50 of 66) with videos related to

Sort By:
Pageof 7
American Journal of Hypertension|October 4, 2021
An Evaluation of Cardiac Health in the Spontaneously Hypertensive Rat Colony: Implications of Evolutionary Driven Increases in Concentric HypertrophyEmma J B Holjak, Iryna Savinova, Victoria L Nelson, et al.
Redox Biology|August 23, 2024
Mitohormesis during advanced stages of Duchenne muscular dystrophy reveals a redox-sensitive creatine pathway that can be enhanced by the mitochondrial-targeting peptide SBT-20Meghan C Hughes, Sofhia V Ramos, Aditya N Brahmbhatt, et al.
Science Translational Medicine|May 19, 2017
Central-acting therapeutics alleviate respiratory weakness caused by heart failure-induced ventilatory overdriveAndrew J Foster, Mathew J Platt, Jason S Huber, et al.
Frontiers in Physiology|July 16, 2024
Cardiomyocyte crosstalk with endothelium modulates cardiac structure, function, and ischemia-reperfusion injury susceptibility through erythropoietinJade P Marrow, Razan Alshamali, Brittany A Edgett, et al.
Chronobiology International|July 25, 2012
Chronomics of pressure overload-induced cardiac hypertrophy in mice reveals altered day/night gene expression and biomarkers of heart diseaseElena V Tsimakouridze, Marty Straume, Peter S Podobed, et al.
CJC Open|June 7, 2024
Better Respiratory Function in Heart Failure Patients With Use of Central-Acting TherapeuticsJulie K K Vishram-Nielsen, Fernando Luis Scolari, Chun-Po Steve Fan, et al.
Journal of the American Society of Nephrology : JASN|January 24, 2016
Nephrin Tyrosine Phosphorylation Is Required to Stabilize and Restore Podocyte Foot Process ArchitectureLaura A New, Claire E Martin, Rizaldy P Scott, et al.
Journal of the American College of Cardiology|May 14, 2011
The primary benefits of angiotensin-converting enzyme inhibition on cardiac remodeling occur during sleep time in murine pressure overload hypertrophyTami A Martino, Nazneen Tata, Jeremy A Simpson, et al.
Circulation. Arrhythmia and Electrophysiology|July 13, 2011
Iron overload decreases CaV1.3-dependent L-type Ca2+ currents leading to bradycardia, altered electrical conduction, and atrial fibrillationRobert A Rose, Michael Sellan, Jeremy A Simpson, et al.
Journal of the American Society of Nephrology : JASN|July 29, 2022
Complementary Nck1/2 Signaling in Podocytes Controls <b></b> Actinin-4-Mediated Actin Organization, Adhesion, and Basement Membrane CompositionClaire E Martin, Noah J Phippen, Ava Keyvani Chahi, et al.
Pageof 7