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Plos One|May 24, 2013
A Kir6.2 pore mutation causes inactivation of ATP-sensitive potassium channels by disrupting PIP2-dependent gatingJeremy D Bushman, Qing Zhou, Show-Ling ShyngFASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|April 11, 2015
A proton current associated with sour taste: distribution and functional propertiesJeremy D Bushman, Wenlei Ye, Emily R LimanThe Journal of Biological Chemistry|December 25, 2009
Characterization and functional restoration of a potassium channel Kir6.2 pore mutation identified in congenital hyperinsulinismJeremy D Bushman, Joel W Gay, Paul Tewson, et al.Pflugers Archiv : European Journal of Physiology|December 6, 2006
Optimized expression vector for ion channel studies in Xenopus oocytes and mammalian cells using alfalfa mosaic virusSrinivasan P Venkatachalan, Jeremy D Bushman, José L Mercado, et al.Oncotarget|August 11, 2018
A k-mer based transcriptomics approach for antisense drug discovery targeting the Ewing's family of tumorsAndrew J Annalora, Shawn O'Neil, Jeremy D Bushman, et al.The Journal of Biological Chemistry|February 6, 2008
Destabilization of ATP-sensitive potassium channel activity by novel KCNJ11 mutations identified in congenital hyperinsulinismYu-Wen Lin, Jeremy D Bushman, Fei-Fei Yan, et al.Proceedings of the National Academy of Sciences of the United States of America|December 3, 2015
The K+ channel KIR2.1 functions in tandem with proton influx to mediate sour taste transductionWenlei Ye, Rui B Chang, Jeremy D Bushman, et al.Pageof 1