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Molecular Cancer Therapeutics|February 16, 2013
Transient exposure to quizartinib mediates sustained inhibition of FLT3 signaling while specifically inducing apoptosis in FLT3-activated leukemia cellsRuwanthi N Gunawardane, Ronald R Nepomuceno, Allison M Rooks, et al.
Journal of Bacteriology|June 7, 2005
A phosphohexomutase from the archaeon Sulfolobus solfataricus is covalently modified by phosphorylation on serineW Keith Ray, Sabrina M Keith, Andrea M DeSantis, et al.
Nature Biotechnology|November 1, 2011
Comprehensive analysis of kinase inhibitor selectivityMindy I Davis, Jeremy P Hunt, Sanna Herrgard, et al.
Chemistry & Biology|November 25, 2010
Activation state-dependent binding of small molecule kinase inhibitors: structural insights from biochemistryLisa M Wodicka, Pietro Ciceri, Mindy I Davis, et al.
Nature Chemical Biology|March 4, 2014
Dual kinase-bromodomain inhibitors for rationally designed polypharmacologyPietro Ciceri, Susanne Müller, Alison O'Mahony, et al.
Nature|April 17, 2012
Validation of ITD mutations in FLT3 as a therapeutic target in human acute myeloid leukaemiaCatherine C Smith, Qi Wang, Chen-Shan Chin, et al.
Nature Biotechnology|January 10, 2008
A quantitative analysis of kinase inhibitor selectivityMazen W Karaman, Sanna Herrgard, Daniel K Treiber, et al.
Synthetic Biology (Oxford, England)|October 26, 2022
Highly-automated, high-throughput replication of yeast-based logic circuit design assessmentsRobert P Goldman, Robert Moseley, Nicholas Roehner, et al.
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