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Scientific Reports|April 26, 2016
PSD95 nanoclusters are postsynaptic building blocks in hippocampus circuitsMatthew J Broadhead, Mathew H Horrocks, Fei Zhu, et al.Nature Neuroscience|June 10, 2008
Evolutionary expansion and anatomical specialization of synapse proteome complexityRichard D Emes, Andrew J Pocklington, Christopher N G Anderson, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|November 13, 2002
SynGAP regulates ERK/MAPK signaling, synaptic plasticity, and learning in the complex with postsynaptic density 95 and NMDA receptorNoboru H Komiyama, Ayako M Watabe, Holly J Carlisle, et al.Nature Neuroscience|December 4, 2012
Evolution of GluN2A/B cytoplasmic domains diversified vertebrate synaptic plasticity and behaviorTomás J Ryan, Maksym V Kopanitsa, Tim Indersmitten, et al.Neuron|May 15, 2012
The subtype of GluN2 C-terminal domain determines the response to excitotoxic insultsMarc-André Martel, Tomás J Ryan, Karen F S Bell, et al.Molecular Brain|November 29, 2014
Human post-mortem synapse proteome integrity screening for proteomic studies of postsynaptic complexesÀlex Bayés, Mark O Collins, Clare M Galtrey, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|September 24, 2010
Neuroligin 1 is dynamically exchanged at postsynaptic sitesInga U Schapitz, Bardo Behrend, Yvonne Pechmann, et al.EMBO Reports|May 15, 2010
The SH3 domain of postsynaptic density 95 mediates inflammatory pain through phosphatidylinositol-3-kinase recruitmentMargaret I Arbuckle, Noboru H Komiyama, Ada Delaney, et al.Biochemical Society Transactions|March 20, 2010
Confirmed rare copy number variants implicate novel genes in schizophreniaGloria W C Tam, Louie N van de Lagemaat, Richard Redon, et al.Elife|July 22, 2017
Pro-death NMDA receptor signaling is promoted by the GluN2B C-terminus independently of Dapk1Jamie McQueen, Tomás J Ryan, Sean McKay, et al.Pageof 17