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American Journal of Physiology. Renal Physiology|October 5, 2006
WNK4 enhances TRPV5-mediated calcium transport: potential role in hypercalciuria of familial hyperkalemic hypertension caused by gene mutation of WNK4Yi Jiang, William B Ferguson, Ji-Bin PengBiochemistry|February 4, 2016
Molecular Modeling of the Structural and Dynamical Changes in Calcium Channel TRPV5 Induced by the African-Specific A563T VariationLingyun Wang, Ross P Holmes, Ji-Bin PengJournal of Biomolecular Structure & Dynamics|September 4, 2018
Modeling the structural and dynamical changes of the epithelial calcium channel TRPV5 caused by the A563T variation based on the structure of TRPV6Lingyun Wang, Ross P Holmes, Ji-Bin PengBiochemical and Biophysical Research Communications|August 30, 2017
The L530R variation associated with recurrent kidney stones impairs the structure and function of TRPV5Lingyun Wang, Ross P Holmes, Ji-Bin PengNews in Physiological Sciences : an International Journal of Physiology Produced Jointly by the International Union of Physiological Sciences and the American Physiological Society|July 19, 2003
Apical entry channels in calcium-transporting epitheliaJi-Bin Peng, Edward M Brown, Matthias A HedigerThe Journal of Physiology|July 19, 2003
Epithelial Ca2+ entry channels: transcellular Ca2+ transport and beyondJi-Bin Peng, Edward M Brown, Matthias A HedigerJournal of Biomolecular Structure & Dynamics|August 11, 2022
Molecular insights into the structural and dynamical changes of calcium channel TRPV6 induced by its interaction with phosphatidylinositol 4,5-bisphosphateLingyun Wang, Ruiqi Cai, Xing-Zhen Chen, et al.Biochemical and Biophysical Research Communications|October 18, 2005
Characterization of a branched-chain amino-acid transporter SBAT1 (SLC6A15) that is expressed in human brainHitomi Takanaga, Bryan Mackenzie, Ji-Bin Peng, et al.The Journal of Biological Chemistry|September 17, 2010
Down-regulation of intestinal apical calcium entry channel TRPV6 by ubiquitin E3 ligase Nedd4-2Wei Zhang, Tao Na, Guojin Wu, et al.American Journal of Physiology. Renal Physiology|October 12, 2012
Disease-causing R1185C mutation of WNK4 disrupts a regulatory mechanism involving calmodulin binding and SGK1 phosphorylation sitesTao Na, Guojin Wu, Wei Zhang, et al.Pageof 5