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BME Frontiers|October 18, 2023
Multimodal Metabolic Imaging Reveals Pigment Reduction and Lipid Accumulation in Metastatic MelanomaHyeon Jeong Lee, Zhicong Chen, Marianne Collard, et al.
Blood|January 24, 2018
Extracellular vesicle-mediated transfer of constitutively active MyD88<sup>L265P</sup> engages MyD88<sup>wt</sup> and activates signalingMateja Manček-Keber, Duško Lainšček, Mojca Benčina, et al.
Nature Methods|October 1, 2025
Giotto Suite: a multiscale and technology-agnostic spatial multiomics analysis ecosystemJiaji G Chen, Joselyn C Chávez-Fuentes, Matthew O'Brien, et al.
Blood|February 26, 2017
Acquired mutations associated with ibrutinib resistance in Waldenström macroglobulinemiaLian Xu, Nicholas Tsakmaklis, Guang Yang, et al.
Journal of Clinical Oncology : Official Journal of the American Society of Clinical Oncology|July 26, 2018
Ibrutinib Monotherapy in Symptomatic, Treatment-Naïve Patients With Waldenström MacroglobulinemiaSteven P Treon, Joshua Gustine, Kirsten Meid, et al.
Blood|June 16, 2016
Transcriptome sequencing reveals a profile that corresponds to genomic variants in Waldenström macroglobulinemiaZachary R Hunter, Lian Xu, Guang Yang, et al.
Blood Advances|January 15, 2020
Expression of the prosurvival kinase HCK requires PAX5 and mutated MYD88 signaling in MYD88-driven B-cell lymphomasXia Liu, Jiaji G Chen, Manit Munshi, et al.
Blood|May 5, 2016
HCK is a survival determinant transactivated by mutated MYD88, and a direct target of ibrutinibGuang Yang, Sara J Buhrlage, Li Tan, et al.
British Journal of Haematology|September 6, 2018
TP53 mutations are associated with mutated MYD88 and CXCR4, and confer an adverse outcome in Waldenström macroglobulinaemiaJoshua N Gustine, Nicholas Tsakmaklis, Maria G Demos, et al.
British Journal of Haematology|July 4, 2019
CXCR4 mutation subtypes impact response and survival outcomes in patients with Waldenström macroglobulinaemia treated with ibrutinibJorge J Castillo, Lian Xu, Joshua N Gustine, et al.
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