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Jil C Tardiff

Showing results (1-10 of 60) with videos related to

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Circulation Research|March 19, 2011
Thin filament mutations: developing an integrative approach to a complex disorderJil C Tardiff
The Journal of Clinical Investigation|June 3, 2006
Cardiac hypertrophy: stressing out the heartJil C Tardiff
Heart Failure Reviews|January 18, 2006
Sarcomeric proteins and familial hypertrophic cardiomyopathy: linking mutations in structural proteins to complex cardiovascular phenotypesJil C Tardiff
American Journal of Physiology. Heart and Circulatory Physiology|December 7, 2010
Abnormal heart rate regulation in murine hearts with familial hypertrophic cardiomyopathy-related cardiac troponin T mutationsJesus Jimenez, Jil C Tardiff
The Journal of General Physiology|January 29, 2021
Complexity in genetic cardiomyopathies and new approaches for mechanism-based precision medicineMichael J Greenberg, Jil C Tardiff
The Journal of Physical Chemistry. B|November 30, 2022
Insights into the Mechanism of the Cardiac Drug Omecamtiv Mecarbil─A Computational StudyAnanya Chakraborti, Jil C Tardiff, Steven D Schwartz
American Journal of Physiology. Heart and Circulatory Physiology|July 5, 2005
Increase in tension-dependent ATP consumption induced by cardiac troponin T mutationMurali Chandra, Matthew L Tschirgi, Jil C Tardiff
The Journal of Physical Chemistry. B|May 6, 2024
Myosin-Catalyzed ATP Hydrolysis in the Presence of Disease-Causing Mutations: Mavacamten as a Way to Repair MechanismAnanya Chakraborti, Jil C Tardiff, Steven D Schwartz
Archives of Biochemistry and Biophysics|February 1, 2014
Allosteric effects of cardiac troponin TNT1 mutations on actomyosin binding: a novel pathogenic mechanism for hypertrophic cardiomyopathyRachel K Moore, Salwa Abdullah, Jil C Tardiff
Biophysical Journal|July 13, 2024
Human cardiac β-myosin powerstroke energetics: Thin filament, Pi displacement, and mutation effectsBai Hei, Jil C Tardiff, Steven D Schwartz
Pageof 6

Showing results (1-10 of 60) with videos related to

Sort By:
Pageof 6
Circulation Research|March 19, 2011
Thin filament mutations: developing an integrative approach to a complex disorderJil C Tardiff
The Journal of Clinical Investigation|June 3, 2006
Cardiac hypertrophy: stressing out the heartJil C Tardiff
Heart Failure Reviews|January 18, 2006
Sarcomeric proteins and familial hypertrophic cardiomyopathy: linking mutations in structural proteins to complex cardiovascular phenotypesJil C Tardiff
American Journal of Physiology. Heart and Circulatory Physiology|December 7, 2010
Abnormal heart rate regulation in murine hearts with familial hypertrophic cardiomyopathy-related cardiac troponin T mutationsJesus Jimenez, Jil C Tardiff
The Journal of General Physiology|January 29, 2021
Complexity in genetic cardiomyopathies and new approaches for mechanism-based precision medicineMichael J Greenberg, Jil C Tardiff
The Journal of Physical Chemistry. B|November 30, 2022
Insights into the Mechanism of the Cardiac Drug Omecamtiv Mecarbil─A Computational StudyAnanya Chakraborti, Jil C Tardiff, Steven D Schwartz
American Journal of Physiology. Heart and Circulatory Physiology|July 5, 2005
Increase in tension-dependent ATP consumption induced by cardiac troponin T mutationMurali Chandra, Matthew L Tschirgi, Jil C Tardiff
The Journal of Physical Chemistry. B|May 6, 2024
Myosin-Catalyzed ATP Hydrolysis in the Presence of Disease-Causing Mutations: Mavacamten as a Way to Repair MechanismAnanya Chakraborti, Jil C Tardiff, Steven D Schwartz
Archives of Biochemistry and Biophysics|February 1, 2014
Allosteric effects of cardiac troponin TNT1 mutations on actomyosin binding: a novel pathogenic mechanism for hypertrophic cardiomyopathyRachel K Moore, Salwa Abdullah, Jil C Tardiff
Biophysical Journal|July 13, 2024
Human cardiac β-myosin powerstroke energetics: Thin filament, Pi displacement, and mutation effectsBai Hei, Jil C Tardiff, Steven D Schwartz
Pageof 6