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Oncotarget|June 17, 2021
Frame-shift mediated reduction of gain-of-function p53 R273H and deletion of the R273H C-terminus in breast cancer cells result in replication-stress sensitivityViola Ellison, George K Annor, Clara Freedman, et al.Current Opinion in Oncology|January 16, 2002
Multiple roles of the tumor suppressor p53Jill Bargonetti, James J ManfrediJournal of Nucleic Acids|August 28, 2010
Differential toxicity of DNA adducts of mitomycin CJill Bargonetti, Elise Champeil, Maria TomaszOncotarget|September 30, 2015
Homozygous mdm2 SNP309 cancer cells with compromised transcriptional elongation at p53 target genes are sensitive to induction of p53-independent cell deathMelissa Rosso, Alla Polotskaia, Jill BargonettiCold Spring Harbor Perspectives in Biology|August 4, 2010
Transcriptional regulation by p53Rachel Beckerman, Carol PrivesSeminars in Cell & Developmental Biology|April 21, 2005
The S phase checkpoint: when the crowd meets at the forkVanesa Gottifredi, Carol PrivesGenes & Development|May 17, 2008
Does control of mutant p53 by Mdm2 complicate cancer therapy?Carol Prives, Eileen WhiteCell Cycle (Georgetown, Tex.)|December 11, 2008
The p21 and PCNA partnership: a new twist for an old plotCarol Prives, Vanesa GottifrediPageof 17